﻿276 The Philippine Journal of Science 1914 



present; however, the intestines were cedematous and adherent 

 as in A-21. 



LUNGS 



More or less pleurisy was present in all 6 cases. The lobes 

 adhered to each other and in some instances to the thoracic wall. 

 In 3 cases, kidney worms were found in the thoracic cavity, but, 

 as the bronchioles in every case contained large numbers of 

 worms belonging to the genus Metastrongylus, it is rather diffi- 

 cult to decide whether the pleurisy was caused by them or by 

 Stephanurus dentatus. 



MICROSCOPICAL EXAMINATION OF INFESTED ORGANS 



The tissue under consideration was fixed in both Zenker's fluid 

 and formalin, and paraffin as well as frozen sections were made. 

 The sections were stained with Ehrlich's acid haematoxylin and by 

 Van Gieson's method, using Ehrlich's acid haematoxylin and 

 picrofuchsin. Sections revealed the following conditions: 



KIDNEYS 



The kidney tissue showed a pronounced passive congestion. 

 The veins and capillaries between the tubules in both coi'tex and 

 medulla were distended with blood. In some instances, a con- 

 siderable amount of congestion was present in the glomeruli. 

 Since the arteries were practically empty, undoubtedly the con- 

 gestion of the glomeruli was due to the enormous passive conges- 

 tion which dammed the blood back in them. 



Parenchymatous degeneration was present in all stages of 

 development and scattered diffusely throughout the cortical and 

 medullary substance. Numerous casts were present in the con- 

 voluting and collecting tubules (Plate II, fig. 2, a). These casts 

 had all the appearances and staining reactions of colloid material, 

 for instance giving a homogeneous orange-red color, when 

 stained with haematoxylin and picrofuchsin. 



Numerous parenchymatous cells were noticed undergoing 

 colloid degeneration. They presented different appearances, 

 depending upon the extent to which the degeneration had taken 

 place. In the earliest stage noticeable, the cell body took a lighter 

 stain and was very finely granular. In the next stage, small 

 spherical droplets, which took an orange stain with haematoxylin 

 and picrofuchsin (Plate II, fig. 3, a), could be noticed scattered 

 throughout the cell body. As the degeneration progressed, the 

 droplets became larger and finally occupied the entire cell body 

 (Plate II, fig. 3, c). Finally, they burst out into the lumen of 

 the tubule, and eventually coalesced to form casts (Plate II, fig. 



