Pd eee 
Por 
Se ae eye ae ee ee ee er freee eee a Se ee Se 
> TEU A Oe ee a aRR EE aera 
sas) N ances ae ates amend chee a has ated se orang | oh i avay poh cy ee te cee tee a la las ee le 
G. F. Barker—Physiological Chemistry. 57 
From this it will be seen that the volume of the liver of the 
cat in a state of health and when fed on meat is almost double 
that of the rabbit at the period of most active digestion ; 
parece, the liver of a large well-fed cat gives not more 
2 the quantity of amyloid matter yielded by the liver of 
a rabbit fed on carrots, bread, and parsley. Saccharine food 
causes the production “of amyloid. matter in the liver much 
more readily than nitrogenous food ; although it isa well es- 
tablished fact that this organ can make the amyloid substance 
from blood-fibrin, from wheat-glutin, or from fresh meat. 
McDonnell claims also to have proved, contrary to the opinion 
of an eminent physiologist, that there is no more amyloid mat- 
ter in the liver of an animal fed on gelatin than there is in the 
liver of a fasting animal. Gelatin therefore, is not trans- 
formed into amyloid matter by the liver. The same is true of 
t. 
(71.) The same year F. Rirrer published* some experiments 
upon the normal conversion of glycogen. His paper begins by 
stating the views at that time held upon this subject. “As to 
its origin he inclines to the belief that it comes from the split- 
ting up of albuminates, rather than from a metamorphosis of 
carbohydrates. Pavy’s assertion of its non-conversion into 
Professor Meissner’s Ee "The method which te pursued 
is as follows: the abdomen of a sete rabbit, firmly held by 
a sha : 
large a liver-fragment i is to be avoided ; and the i Set Be 
to use scissors in cutting it fine, holding it over the boilin 
on heating to eae no separation of cuprous im took place, 
a few drops of a freshly prepared soiution of potassic ferricy- 
absence 
_ anid were added ; a pure yellow precipitate proved the 
* Zeitschr. f. rat. Med., III, xxiv, 65, 1865. 
