282 Veterinary Medicine. 



ing animals, quick pulse, congested mucosae, diarrhoea, cramps, 

 weakness and paresis, and if the patient survives, anaemia and 

 emaciation. 



Lesions. The mucosa of mouth, gullet, stomach and perhaps 

 duodenum is white, opaque, hard, corrugated, leathery, slough- 

 ing, or ulcerated. Congestion is well marked. Strictures may- 

 appear in chronic cases. 



Treatment. Give tepid water and tickle the fauces. Use 

 white of egg or milk freely and mucilaginous agents. Tannic 

 acid, or carbonate of soda are antidotal by tending to precipitate 

 insoluble compounds. 



POISONING BY SILVER. 



Toxic doses of silver come mostly from materials used in the 

 arts. The photographer uses chiefly the nitrate, iodide, bromide, 

 cyanide and chloride. Taken into the stomach the silver salts 

 are less poisonous because they are largely precipitated as in- 

 soluble chloride or albuminate. The chloride and albuminate 

 are, however, soluble in solutions of alkaline chlorides and hence 

 even they may poison. 



Symptoms. Colic, emesis in vomiting animals the vomited 

 matters blackening in the light, diarrhoea, great muscular weak- 

 ness, paresis, weak clonic spasms, and disturbed respiration. 

 The nervous symptoms are very prominent (Rouget and Curci). 

 Chronic poisoning produces emaciation and fatty degeneration 

 of liver, kidneys and muscles f Bogoslowsky). 



Lesions. Patches, of congestion and of white corrosion on the 

 buccal, oesophageal and gastric mucous membrane, the presence 

 of the curdy white chloride of silver adherent to the gastric mu- 

 cosa. In chronic cases the visible mucosae and white skin may 

 have a slaty color. 



Treatment. Emetics in vomiting animals. White of egg, 

 common salt largely diluted and followed by milk as antidotal, 

 demulcent and nutritive agent. 



