GLYCOSURIA IN CATTLE. 



Accompaniment of parturition fever and apoplexy. Essential glycosuria. 

 Dense saccharine urine, passed often, congested mucosae, emaciation. Le- 

 sions uncertain. In parturition fever and apoplexy the congested medulla 

 is the reasonable starting point. Toxic glycosuria. Treatment: addressed 

 to the primary disease or poison ; otherwise treat as in the horse. 



In cattle this has been observed as a symptomatic affection in 

 connection with parturition fever or apoplexy (Nocard, St. Cyr, 

 Violet). One case of essential mellituria has been recorded by 

 Darbas. 



Symptoms. In the last mentioned case in a work ox, the ani- 

 mal, when at work, would stop every five or ten minutes to 

 urinate, passing a small quantity of amber colored urine of a high 

 density and containing a large amount of glucose. The con- 

 junctiva was pink, the animal considerably emaciated, and rest 

 and generous feeding brought about no improvement, so that 

 the subject was finally sent to the butcher to anticipate a natural 

 death. 



The lesions in this case are altogether hypothetical. The red 

 eyes might imply congestion of the encephalon (medulla), but 

 the redness might be caused by active disease in the liver, pan- 

 creas or kidney. The failure to notice jaundice does not indi- 

 cate a healthy liver, as some of the most fatal diseases of that 

 organ are unattended by icterus. The frequent emission of 

 urine in small amounts would imply irritation in kidneys or blad- 

 der, from which the glycogenic stimulus may have started. In 

 the absence of any more definite evidence of disease in other or- 

 gans, it is, however, more probable that the fundamental disorder 

 resided in the liver, the great glycogenic factor of the body. 



In parturition fever, the presumption is in favor of considering 

 the congested medulla as the starting point of the disease, yet in 

 view of the manifest paralysis of stomach and bowels, it is not 

 improbable that the vascular congestion and paralysis of the 

 chylo-poietic viscera constituted the initial step in the morbid 

 process, while the glycogenesis was the result of a reflex opera- 

 tion on the liver. 

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