Hemorrhagic Infarction of the Spleen. 575 



toms of such primary diseases. In those resulting from trau- 

 matic injury, bruises, swellings or wounds, cutaneous or subcuta- 

 neous, there will often be suggestive features. In the more purely 

 idiopathic cases symptoms are only shown when the lesions are 

 extensive and acute. In oxen, Cruzel has noted the initial chill, 

 followed by disturbance of the respiration, more or less hyper- 

 thermia, and a swelling of the left flank and hypochondrium in 

 the absence of tympany of the rumen. The nature of this swell- 

 ing is the most characteristic feature, as it gives a flat instead of 

 a drumlike sound on percussion, and does not bulge outward and 

 downward over the whole left side of the abdomen, pit on pres- 

 sure, nor crepitate uniformly all over from fermentation, as in 

 overloading of the stomach. 



If abscess should form, chills and high febrile reaction are 

 marked symptoms. In vomiting animals, anorexia, nausea, 

 vomiting, constipation, and even diarrhoea may appear. 



Prognosis. Unless in extreme cases and those due to trauma- 

 tism or infection, the result of splenitis is usually favorable. 

 • Treatment would consist in depletion from the portal system 

 and spleen by rectal injections, and laxatives which like castor 

 oil, will operate without extensive absorption. Cold water or 

 ice is applied to the left flank and induction currents of electricity 

 may also be resorted to. General blood letting is strongly 

 advised by Cruzel, and Friedberger and Frohner. .In infective 

 cases quinia, salicylates, salol, and the sulphites, or iodides would 

 be indicated. 



HEMORRHAGIC INFARCTION OF THE SPLEEN. 



In congestive conditions. Absence of free capillary anastomosis and con- 

 traction, absence of valves in splenic veins. Embolism of splenic artery. 

 Clots in pulp spaces. Wedge shaped infarcts, first black, later yellow, later 

 caseated, or cicatrized. Abscess. Prognosis good in non-infective forms. 

 Treatment as for hypersemia, or infection, or both. 



This condition appears in hypersemia, hypertrophy, splenitis, 

 and splenic infection and largely because the structure 

 and circulation in the organ conduce to such trouble. The 



