156 



NEI'ERINARV PATHOLOGY. 



clency to excessive exudation. A weak heart is conducive to 

 excessive exudation, e. g., inflammatory oedema. Animals pos- 

 sessinq- dilute blood (hydremia) are predisposed to excessive 

 fluid exudation. The leucocytic amoeboid movement may be tem- 

 porarily suspended, or it may be increased during inflamma- 

 tion, resulting in an absence or in an excessive number of 

 leucocytes in the exudate. In animals having a clean close 

 build the exudate is not so extensive as it is in tliose animals 

 of a loose flabb}- make-up. 



c. The location and tissue affected. Exudation is in (hrcct pro- 

 portion til the vascularity- and density of the tissue. Inflam- 

 mation in compact bonv tissue or beneath dense facia, liga- 



ments or tendons is accompanied b^' a limited exudate. In- 

 flammation of the cutaneous structure is usuallv associated 

 with excessive exudation, which accumulates in the subcutane- 

 ous areolar tissue. Inflamn:atifjn of serous and mucous mem- 

 branes is accompanied with exudation which may in part be 

 discharged upon the surface Init is usualh- accumulated in the 

 substructures. 



4. Significance of the exudate. — The significance of the exu- 

 date has liad various interjiretations. \'irchow considered that 

 the irritation producing the inflammation resulted in increased 

 cellular acti^'ity in the injured area and that the exudate sup- 

 plied increased nourishment to the area in which there was 

 an excessive metabolism, fjthers have attributed to the exudate 

 the "flushing out" of the injured area thus mechanically carrying 



