•jQ Veterinary Medicine. 



and, if they survive the resulting fever, would repeat the treat- 

 ment after the hyperthermia has ceased. No satisfactory treat- 

 ment of the disease has been made. An open air life, a generous 

 diet, and a course of iron, and bitters will, however, be of use in 

 serving to improve health, digestion and vigor, to solicit a better 

 production of red globules, and to enable the patient to survive 

 the period of anaemia, prostration and debility. Antiseptics like 

 quinia, the sulphites and the iodides might be used as adjuncts. 



PNEUMO-ENTERITIS IN SHEEP : HEMORRHAGIC 

 SEPTICEMIA : SWINE PLAGUE. 



Historic note. Microbiology : ovoid bacterium, motile, with polar stain, 

 non-liquefying, chains, grows freely on culture medja. Pathogenic to sheep, 

 goats, dogs, hens, rodents, calf, ass. Views of Lignieres, Lienaux, and 

 Conte. Symptoms : Acute form in young ; hyperthermia, rapid pulse, 

 troubled breathing, dulness, prostration, sopor, anorexia, congested petechi- 

 ated mucosae, offensive diarrhoea, emaciation, wheezing, cough, riles, crepi- 

 tus, percussion flatness, abortion. Death in 6 hours to 3 days. Sub-acute 

 form in mature : symptoms moderate, recoveries the rule. Lesions : foetid 

 carcass, blood staining of skin and organs, exudates, petechise, swollen 

 congested lymph glands, peritoneal exudate, congested liver and spleen, 

 gastro-enteritis, pleural effusion, lobular and peribronchial exudates, casea- 

 tion, congested womb, placenta and brain, bacterium in lesions. Preven- 

 tion : isolation, disinfection, secretions, manure, drainage, exclude tame 

 and wild animals. Disinfectants. 



Among the different forms of haemorrhagic septicaemia in sheep, 

 that observed by Galtier in 1889 in Basses Alpes, and later else- 

 where in southern and western France and in Algiers, must be 

 specially noted. It seems to be the same affection studied later 

 by Lienaux, Conte, Besuoit and Cuille and which prevailed from 

 Tarn in the south of France, to Vendee in the west, and Somme 

 in the north. 



Microbiology. The pathogenic factor found in the lesions was 

 an ovoid bacterium, a little larger than that of fowl cholera, mo- 

 tile, non-liquefying, with polar staining, and often showing in 

 short chains of two or three joined end to end. It grows easily 

 and abundantly in all common culture media, even on potato 

 which fails to propagate the cocco-bacillus of Lignieres. This, 



