LAMENESS, FROM VARIOUS CAUSES. 367 
dead. We infer that necrosis took place, and the extensor ten- 
dons lost their at ‘achment, which accounted for his breaking down. 
Prof. VARNELL lately consulted Dr. HARLEY, of London, in 
reference to this formidable malady, which is said to have been 
almost unknown in England until November, 1859, when several 
cases occurred among some horses the property of Mr. Champion 
Calcot, near Reading. Three of this gentleman’s animals had 
died, a fourth was not expected to live many days, and two others 
were laboring under the same disease, in a less acute form. The 
most singular feature of the disease, as it appeared on the prem- 
ises of Mr. Calcot, was, that it did not appear to owe its origin 
to any perceivable cause, and it was not known to be prevalent in 
any other part of England. Still, our opinion is that it does 
exist in that country, only, not being suspected, is not sought for. 
It prevails very extensively among horses brought to Ohio, and 
hundreds of horses have been purchased for the American Gov- 
ernment having more or less enlargement of the lower jaw, yet 
we do not believe that either seller or purchaser suspected the 
same, ‘This arises from a lack of knowledge on the subject, and 
the same remarks, as far as our ignorance of the subject is con- 
ceened, may apply to England. 
In answer to questions put to Mr. Calcot on the subject of man- 
agement, etc., he contends that the affected animals had plenty 
of exercise. There were no chemical factories or works in the 
vicinity. One horse affected was purchased at a distance; the 
rest had been bred on his farm. The sires and dams all appeared 
free from the disease, and they were not all got by the same sire. 
The food consisted of good grass, hay, pollard, oatmeal, and roots. 
Other horses were on the farm, fed in the same manner as the 
sffected ones, yet they had no symptoms of the disease. (Perhaps 
\hey will show symptoms when the jaws shall be examined. They 
tnay have the disease, yet not be lame nor apparently ailing.) 
‘ir. Calcot never saw a case of the kind before. 
From the above testimony, we may infer that the causes of this 
affection are not yet discovered in Englaxd; but our impression 
is that overfeeding has a good deal to do in the production of 
the malady. It appears to me that this disease, as I have already 
itated, is one of malnutrition, or defective nourishment; hence 
tay be considered as a scorbutic affection, like that affecting the 
human subject, which is known to be the result of faulty nutri- 
