Draft op Law for the Prevention of Infectious Axemia. 59 



pasture, the patient stays separated from the rest of 

 the herd, and does not respond to whipping. In the 

 stable, the affected animal stands in one place lowering 

 its head. If it is forced to move, it shows signs of 

 fatigue and tendancy to perspire. Its locomotion is greatly 

 interfered with. It does not react to pressing applied at 

 its hind quarters. It lacks the power of lifting its tail. 

 It is not inclined to get rid of flies, if they swarm 

 about its body. 



The pulse increases to 50-80-ca 100 per minute. 

 Palpitation becomes forcible and can readily be felt on 

 the chest. The heart action can even be seen from 

 without. Pulsation of the jugular vein can also be 

 noticed. 



The heart sound is often muffled or intermittent or 

 doubled. 



Oedema is also a frequent symptom. It appears in 

 the lower part of the head and neck, chest, belly, distal 

 end of the extremities and external genital organ. In 

 the incipient stage, the visible mucous membranes flushes, 

 but, when anemia sets in, they gradually become yellowish 

 white or grayish. Often dot-like small petechiae are seen. 



The blood becomes thin and watery. The red blood 

 corpuscles decrease extremely in number. If the blood 

 is left to stand in a test-tube the corpuscular layer at 

 the bottom does not reach half the thickness of that 

 of normal blood. 



Excepting during the febrile periods, the appetite 

 is not impaired. The faeces do not show anything 

 abnormal. The patient is liable to emaciation. 



In acute cases, hypertrophied spleen can be felt upon 

 rectal examination, while during the interfebrile periods 

 and towards the last stage of anemia the spleen does 

 not show enlargement at all. 



