MORBID ANATOMY 247 



examined in fresh condition or in sections of tissue fixed in 

 Miiller's fluid the engorgement of the bile canaliculi is seen. 

 The bile stasis may occur over a portion or a whole of the 

 lobule. The gall bladder contains usually an abnormal 

 quantity of changed bile. It is thick and often semi-solid in 

 consistence holding in suspension many ffocculi. It imparts a 

 deep yellowish tinge to all articles coming in contact with it. 

 Owing to the mucus which is present it is quite viscid. 



The changes in the kidneys vary. If death occurs early 

 they are usually enlarged and uniformly darker than normal 

 throughout. The capillaries are distended with red corpuscles. 

 Parenchymatous and fatty degeneration are not common 

 although occasionally present in the epithelium of the tubules. 

 The pelvis is often sprinkled with ecchymoses. 



The bladder may contain ecchymoses. 



It is important to note that throughout the kidneys, liver 

 and spleen pigment may be more or less abundant. 



In the digestive tract the lesions of this disease consist 

 largely of congestion of the mucosa especially in lines corres- 

 ponding to the summits of the folds of the mucous membrane. 

 It is more marked in the caecum and rectum than in the colon. 

 The caecum and less often the rectum contain dr}', hard fecal 

 balls. In some cases in this system lesions are not observable. 

 In the older writings much emphasis is placed on certain 

 lesions, mostly nodular or abrasions, in the digestive tract. 

 Smith has shown, however, that most of these are due to 

 animal parasites and have no relation to Texas fever. 



As already stated Texas fever is a disease of the blood 

 and consequently it is in this fluid that the most constant and 

 morbid changes occur. They are characterized by the blood 

 becoming thin and watery with a destruction of the red blood 

 corpuscles. In some of the cases the loss of corpuscles is 

 rapid and continuous until death or convalescence, while 

 in others there is a marked oscillation between destruction and 

 regeneration. In some animals the loss is not continuous, but 

 the course of the disease is marked by periods of rapid blood 

 destruction and periods of rest, or, as it were, where the blood 

 destruction was holding its own. These points are best illus- 



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