398 DISEASES OF THE HORSE’S FOOT 
ridden, and after about seven or eight minutes she began 
to go lame in a hind-limb. Her lameness got rapidly worse 
as she was being ridden, and within a quarter of mile of 
her first showing lameness, she dropped and carried the 
lame foot in a way that suggested a badly fractured 
pastern. There was no recognisable disease in the limb 
to account for this lameness. 
‘TI divided the posterior tibial nerve, and she went back 
to work moving sound, and continued to work sound up to 
her death from one of the regularly fatal bowel lesions— 
twist or rupture. 
‘She worked nearly two years after unnerving, and 
developed the usual thickening at the coronet.’* 
2. ‘The subject of this note was a chestnut mare, nine 
years old, and used for omnibus work. 
‘ History.—For about two months the mare was lame 
on the off fore-leg, and in spite of treatment the con- 
dition became steadily worse. The off fore-foot was 
rather long and narrow, and the fetlock-joint was inclined 
to be bowed outwards, but the degree of lameness was out 
of proportion to these defects, and the diagnosis was 
obscure. 
‘Median neurectomy was performed on May 10, 1902, 
and reduced the lameness to about half of what it was 
before. On June 5 ulnar neurectomy was performed, with 
the result that the mare became sound, and went to work 
three weeks later. She continued to work soundly and 
well, being inspected from time to time. 
“During February of 1903 the coronet began to enlarge 
in front and slightly to the outer side, and gradually 
a ridge of bone grew down from the coronet to the toe. 
The case, in fact, became a typical one of so-called “‘ buttress 
foot,” which my friend Mr. Willis has described as diagnostic 
of disease of the pyramidal process of the pedal bone. 
Meanwhile the swelling of the coronet, which appeared to 
be mainly composed of fibrous tissue, increased in size, 
* W. Willis, M.R.C.V.S., Journal of Comparative Pathology and 
Therapeutics, vol. xv., p. 366. 
