46 DISEASES OF DOMESTICATED BIRDS 
atin and not on potato. Milk is coagulated and indol formed. Gel- 
atin is not liquefied. 
Pathogenicity. The mouse is very susceptible to inoculation but 
the white mouse will not contract the infection by ingestion. Birds 
of the order passeres succumb rapidly to intramuscular injection 
of culture. The duck and fowl are insusceptible. Numerous spe- 
cies of swans, geese and ducks in contact with the diseased birds, 
escaped infection. 
Symptoms. The birds remain in a squatting position, and show 
the usual manifestations of sickness exhibited by those affected with 
a septicemia. 
Morbid anatomy. The subjects are generally in good condi- 
tion. The muscles offer nothing special and the heart appears nor- 
mal. Commonly, active congestion is observed in the lungs. Some- 
times one or two caseous foci are present, which are easily enucle- 
ated. The liver is enlarged, dark in color and frequently shows 
small whitish spots. The spleen is sometimes slightly engorged, 
sometimes slightly increased in size. The intestine always shows 
diarrhea with contents yellowish or yellowish green in color. The 
intestines do not show marked injection of the blood vessels. No- 
dules the size of a small pea are frequently found in the abdominal 
cavity in the vicinity of the vertebral column. 
HEMORRHAGIC SEPTICEMIA OF THE SWAN 
Fiorentini observed a septicemic infection among swans in the 
zoological gardens of Milan. 
Morbid anatomy. In young birds that have died quickly there 
is an edematous infiltration of the lungs with ecchymoses on the 
serous membranes. There is also a slight hyperemia of the intes- 
tinal mucosa, dark blood clots in the heart cavity and cloudy degen- 
eration of the liver cells. In older birds that have died after several 
days sickness there are more severe lesions. The lungs show fibrous 
pneumonia in the stage of gray hepatization. The upper lobes of 
the liver show a grayish yellow exudate composed mostly of lym- 
phoid elements, besides marked thickening of Glisson’s capsule. 
The hepatic tissue shows a marked infiltration with red blood cells 
in the spaces between the liver cells. In numerous places in the 
liver there are yellowish zones caused by the fatty degeneration of the 
cellular elements. In the intestine there is slight hyperemia but 
no exudate nor swelling. On the serous membranes there are numer- 
