52 SPECIFIC OPHTHALMIA. 
to gain this end, but the power of the levator of the upper lid causes 
the eye to assume somewhat of a three-cornered aspect. 
It is always desirable to recognize the animal which may be or may 
have been liable to so fearful an affection. 
One symptom of having experienced an at- 
tack is discovered on the margin of the trans- 
parent cornea. The inflammation extends from 
the circumference to the center. The margin 
of the transparent ball is generally the last 
place it quits; here it frequently leaves an 
irregular line of opacity altogether different 
to and distinct from the evenly-clouded indica- 
AN Eve pispLavine Tim Ravaces tion of the cornea’s junction with the sclerotic, 
fie see Ce earwauts which last is natural development. 
Nevertheless, the internal structure best display the ravages of specific 
ophthalmia; it is upon these the terrible scourge exhausts its strength. 
The eye becomes cloudy; loses its liquid appearance; the black bodies 
attached to the edges of the pupillary opening either fall or seem about 
to leave their natural situation. The pupil becomes turbid, then white; 
the iris grows light in color, and at last remains stationary, having pre- 
viously been morbidly active. The whiteness of the pupil grows more 
and more confirmed, and every part grows opaque; by this circumstance, 
the total cataract, arising from specific oph- 
thalmia, is frequently to be challenged, The 
lens, moreover, is often driven, by the force of 
the disease, from its position; it lodges against 
the inner surface of the globe. Very common 
is a torn orragged state of the pupil witnessed, 
as was stated, during the intensity of the at- 
tack, for the iris contracts to exclude the light; 
ce a ee oer remaining thus for any period, it becomes at- 
tached to the capsule of the lens; when the 
disease mitigates, it often rends its own structure by its efforts to expand. 
Should those efforts prove unavailing, the pupillary opening, as some- 
times happens, is lost forever. 
In the previous description of disorder, no mention has been made of 
the cartilago nictitans, or haw, or third eyelid, as it has been called. 
This thin body is very active, and resides at the inner corner of the eye; 
of course, in a disease under which the eye is pained by light, the haw 
is protruded to the utmost. In ophthalmia, however, it is covered by 
an inflamed membrane, and though in health its movements are so 
rapid that it may easily escape notice, yet in this disease it lies before 
