Brencho-Pneumonia from Inhalation. 299 



tubes, but also by the traumas effected by them on the mucosa 

 and the active invasion of the wounds by the swarming micro- 

 organisms. 



The cough is hard, dry and deep, becoming later soft, loose 

 and mucous, but always short and painful. The breathing is 

 accelerated, often reaching 30 or 60 per minute and with per- 

 haps a slight delay between inspiration and expiration, the 

 pulse rises to 80 or 100, and the temperature to 104° to 106° F. 



The difficulty of breathing rapidly encreases being accompanied 

 by coarse mucous r^les and a painful, rattling, suffocative cough. 

 Early in the disease the expired air has a heavy, offensive odor 

 and this goes on encreasing to a marked foetor. The nasal dis- 

 charge, at first serous, or mucous, becomes rapidly thick and pu- 

 rulent, having a yellow tint from escape of blood globules, and 

 then brown from the necrotic products. It assumes a foetid odor 

 at an early stage and becomes more and more offensive as putre- 

 faction and gangrene progress. It may even show particles of 

 the inhaled alimentary matters, coughed up, or debris of disinte- 

 grated lung tissue. 



The case is marked by early and extreme prostration and de- 

 bility, there is a complete loss of life and spirit, a growing stupor, 

 drooping ears and eyelids, indisposition to move, and a generally 

 hopeless appearance. 



Percussion detects in both lungs, and especially in the lower 

 third and anteriorly, limited areas of dullness, — representing the 

 consolidated lobulettes, sometimes a lower flattened area termin- 

 ating above at a horizontal line, and again zones of tympanitic 

 resonance indicating emphysema or the formation of cavities. 



On auscultation the bronchi in the upper part of the lung give 

 coarse mucous rdles, there may be crepitation around the con- 

 solidated patches, with mucous or sibilant riles, or heart or ab- 

 dominal sounds transmitted through their centres. This may be 

 modified at times by the amphoric sound, or, in advanced stages, 

 by splashing or metallic tinkling from the hydro-pneumothorax. 



In the last stages the dyspnoea becomes extreme, the animal 

 is profoundly depressed by the toxin poisoning, he may be sunk 

 in a stupor, and petechise on the visible mucosae may betray the 

 general septicaemia. A profuse and foetid diarrhoea may be 

 among the latest phenomena. 



