Inheritance of Acquired Characters 251 
2. Appearance of epilepsy also in animals born of parents 
which had been rendered epileptic by section of the sciatic 
nerve. 
3. A change in the shape of the ear in animals born of 
parents in which such a change was the effect of a division 
of the cervical sympathetic nerve. 
4. Partial closure of the eyelids in animals born of parents 
in which that state of the eyelids had been caused either by 
section of the cervical sympathetic nerve, or the removal of 
the superior cervical ganglion. 
5. Exophthalmia in animals born of parents in which an 
injury to the restiform body had produced that protrusion of 
the eyeball. This interesting fact I have witnessed a good 
many times, and seen the transmission of the morbid state of 
the eye continue through four generations. In these animals 
modified by heredity, the two eyes generally protruded, 
although in the parents usually only one showed exophthalmia, 
the lesion having been made in most cases only on one of 
the corpora restiformia. 
6. Hematoma and dry gangrene of the ears in animals 
born of parents in which these ear alterations had been caused 
by an injury to the restiform body near the nib of the calamus. 
7. Absence of two toes out of the three of the hind-leg, and 
sometimes of the three, in animals whose parents had eaten 
up their hind-leg toes, which had become anzesthetic from a 
section of the sciatic nerve alone, or of that nerve and also of 
the crural. Sometimes, instead of complete absence of 
the toes, only a part of one or two or three was missing in the 
young, although in the parent not only the toes, but the 
whole foot was absent (partly eaten off, partly destroyed by 
inflammation, ulceration, or gangrene). 
8. Appearance of various morbid states of the skin and 
hair of the neck and face in animals born of parents having 
had similar alterations in the same parts as effects of an 
injury to the sciatic nerve.” 
