THE INHERITANCE OF FAMILY TRAITS 155 



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Fig, 132. — Pedigree of multiple telangiectasis. I, 1, is an English woman 

 who was subject to epistaxis (nosebleed) and had red spots on her face; her 

 daughter, II, 2, 60 years old, has a number of bright red angiomata distributed 

 over face, ears, Ups, tongue, mucous membrane of mouth, and inner surface of 

 all 4 eyelids. During last 6 years has had reciurent attacks of epistaxis. 

 By her first husband she had a son and 8 grandchildren of whom 1 

 suffers from epistaxis. By her second husband she had 8 children of whom 

 III, 3, has had epistaxis since 8 years and 2 small "spider naevi" on left 

 cheek and has a child of 11 who suffers from epistaxis; III, 5, has nose- 

 bleed and 3 small spots on cheek; his son is normal as yet; III, 11, has epis- 

 taxis; III, 16, has sHght attacks of epistaxis but no spots visible. Weber, 1907. 



scendants of the early settlers of Sullivan Co., Pennsylvania, 

 occur "nine-day bleeders." "After the wound is received, 

 instead of heahng a sort of core, of very dark color, composed 

 mostly of coagulated blood forms in the wound, which in 

 about nine days opens, and the blood begins to flow as if 

 from a freshly severed artery. It usually continues to bleed 

 about two weeks, or until the patient is thoroughly ex- 

 hausted, when the "core" falls out and the wound heals. 

 Binding up the wound does no good. The only death known 

 to have occurred through bleeding is supposed to have been 

 caused by binding the wound hghtly to stay the flow of 

 blood." 



That hemophiha has an hereditary basis is generally con- 

 ceded and the conclusion would not be weakened were a 

 specific hemophilia germ some day demonstrated. The par- 

 ticular method of inheritance is well illustrated by Fig. 133 

 of the Sullivan County strain. The males alone are af- 



