CELLULITIS 27 



3. Necrosis, likely to be seen in all forms of cellulitis. 

 Mortification of the diseased tissues is mainly observed in 

 septic, subfacial, intermuscular, perichondreal cellulitis, as 

 poll evil, fistula of tbe witliers, quittor,. Septicaemia and 

 pysemia are often sequels. 



4. Encapsulation jof tbe abscess. Tbe abscess becomes 

 enclosed by a capsule, so to speak, and remains dormant ; 

 most frequently seen in intermuscular cellulitis. A common 

 example is tbe shoulder abscess in the mastoido humeralis of 

 the horse. By and by the capsule gives way and another cel- 

 lulitis attack takes place. This possibly explains the inter- 

 mittent attacks of diffused subcutaneous cellulitis (lymphan- 

 gitis, Monday morning disease, milk leg) so often seen in the 

 liind legs of horses. 



5. Chronic enduration. Sclerosis is often seen after 

 repeated attacks of diffused septic cellulitis in hind legs of 

 horses, representing a chronic connective tissue proliferation ' 

 and leading to enormous thickening of the skin (elephantiasis). 

 Such a chronic connective tissue proliferation involves also 

 subfacial, intermuscular, perichondreal tissues. 



Give the treatment of infectious cellulitis. 



As long as the presence of an abscess is not established, 

 tot antiseptic sponging, irrigations and fomentations must be 

 employed. As soon as possible the abscess should be opened, 

 sloughs removed and the cavity irrigated -with antiseptics and 

 thorough drainage provided. The acute cellulitis of the 

 torse's hind legs is combatted by purgatives. The best 

 treatment of to-day consists in the hypodermic use of are- 

 coline or intravenous use of coUargolum with an external 

 camphor treatment of the leg. 



