ejected from the red cell. The red cell itself was neither 

 enlarged nor distorted as often happens in the case of 

 reptilian erythrocytes when infested by haemogregarines. 



Double infection was common in one of the films examined 

 and even triple infection was seen. In all observed cases 

 of double infection the two parasites were lying side by 

 side either parallel to each other or forming a narrow 

 open V. No schizogonic stages were met with. The films 

 were taken from the heart blood and were stained by 

 Giemsa. The type slide of this parasite for which the 

 name Haemogreyarina (Lankesterella) hylae is proposed, 

 has been presented to the Trustees of the Australian 

 Museum, Sydney, co-types being retained in the Bureau 

 of Microbiology, Sydney. 



The finding of the haemogregarine so commonly in Hyla 

 caerulea but not in H. aurea calls for remark. The two 

 frogs are commonly found together in swamps, but, while 

 H. aurea is essentially a swamp inhabiting species, H. 

 caerulea is very frequently arboreal in its habits. The 

 occurrence of the haemogregarine in one of these often 

 associated species and not in the other, suggests either that 

 the infection by the protozoon is specific in character for 

 the one frog or that its alternative host has access to H. 

 caerulea and not to H. aurea, which latter view seems to 

 us unlikely. The haemogregarines of certain frogs, Rana 

 h-igriua ami li. Iiexydtrtyhi, are stated by Patton 1 to be 

 transmitted by a leech. The same probably holds good for 

 the above described parasite. 



In addition to haematozoa, we have examined frogs for 

 intestinal and other protozoa. Though not in any sense 

 blood parasites, yet we consider it worth while to record 

 the results here :— 



1 W. S. Patton, Parasitology, i, p. 319, 1908. 



