of the substance, but on the clateny tae narcotic effect increased he 
effect in general — the sum of. ie toxic and ‘narcotic ee 
and of lavender oil terpenes killed the same » bacteria. see 5 hours. 
I. R. € eomiees has published an article on the ialiag of. nia ‘oil und 
essential oils, in’ which he endeavours to further ascertain the causes of the p 
brought about by the oils in question. It is easily possible that eucalyptus re} 
has given rise to symptoms of poisoning®) when taken in greater quantities, 
adulterated with products containing phellandrene, as oil of Eucalyptus A 
Labill., and similar eucalyptus species *). ‘The eucalyptus oil used in Bie 
but no phellandrene. Since poe reckons the chellandreties A antaiieged 
amongst the convulsion-producing substances, Spinner believes that the 
in eucalyptus oil glso can be the toxic ingredient. Physiological exper 
this terpene appedring often in nature’), have up to the present ‘been only 
by Fromm and Hildebrandt’). The authors establish that the hydratio 
landrene”’) to phellandrene glucuronic acid takes place in the body. Further, 
'to Spinner, the phenol tasmanol, found by Smith*®) in several eucalyptus 
(EH. linearis and E. Risdoni), may have caused the poisonous Beied on of the ¢ 
the cases mentioned. _ (atl si 
and terpene contents. Erben made the following classification: — 
f 
_ Convulsion- -producing are:— Camphor and the oils of Thuja 6 rina i) 
Hoopla ae ee Paden ieee es Motes Pasthealeine 
virgimana, L., J. Sabina, L., Mentha Pulegium, L., Hedeoma puleaiitesds ), Pe 
europeum, L., Sassafras officinale, Nees, Apium ‘Petroselinum, L. Rey e 
. &, < : y ! Bs 3 
1) Cf. also Reports October 1908, 131; October 1907, 125. — %) Cf. Report at 4k 
12 (1919), 151, 152. — 4) Deutsche Med. Wochenschr. 46 (1920), 389. — De Cf. P. Gait 
fennel oil. — 4) eonscke. t physiol. Chem: 33 (1901), 592; Report April 1902, , 84, Ni ( 
of phellandrene were unknown at that time. — 1°) Cf. is 1916, ae ss ee. 
