328 Edwin F. Hopkins 



Indeed, this blighting may take place when the flower is still in the bud 

 and prevent it from opening. Such a typical bud blight is reproduced 

 in figure 29. 



On the stalks 



While the lesions on the stalks are still small, they have much the same 

 appearance as those on the leaves except that they are more elongate 

 and more depressed. They are of a light brownish color in the center, 

 and are surrounded by a water-soaked area. Older lesions near the base 

 of the stalk appear as dark brown patches and often bear sclerotia on 

 their surfaces, while those higher on the stalk, usually originating in the 

 leaf axils, are grayish white and are covered with conidiophores. Both 

 this blanching effect and the conidial layer are well illustrated in 

 figure 30. 



The extension of the lesion through the stem causes the latter to weaken 

 at the point of attack and break over. If the lesion is near the base, 

 the whole plant topples over; if it is higher up, the flower droops. 



ETIOLOGY 



Nomenclature 



The tulip disease under discussion is caused by Botrytis Tulipae (Libert) 

 comb. nov. A sclerotial form, referred to the form genus Sclerotium by 

 Madame Libert, in 1830, belongs to this species. She called it Sclerotium 

 Tulipae and was the first to describe it (Crypt. Ard., no. 36). As 

 previously mentioned, she apparently did not observe the conidial form. 

 Her original description, which the writer has not been able to see, is 

 taken from Klebahn (1907:5) who quotes it as follows: 



Sclerotium Tulipae, N. Sparsum, adnatum, parvum, ovale, pallide fuscum, laeve, demum 

 nigrum, rugosum, intus album. Ad caules, pericarpia et semina Tulipae Gesnerianae. 

 Autumno. 



In 1836 the species was again designated as Sclerotium Tulipae by 

 Weinmann (1836:647). Sometime between 1841 and 1859 it was once 

 more described in connection with an herbarium specimen, this time 

 by Westendorp. He called it Sclerotium entogenum (Herb, crypt. Belg., 

 no. 827). Finally Cavara (1888) described the fungus more completely, 



