986 



SCIENCE. 



[X. S. Vol. XXI. Xo. 548. 



SOCIETIES AND ACADEMIES. 



THE SOCIETY FOR EXPERIMENTAL BIOLOGY AND 

 MEDICINE. 



The twelfth meeting of the Society for Ex- 

 perimental Biology and Medicine was held in 

 the laboratory of clinical pathology of the Cor- 

 nell University Medical College, on Wednes- 

 day evening, May 24. The Vice-President, 

 Edward K. Dunham, was in the chair. 



Members present. — Atkinson, Auer, Brooks, 

 Burton-Opitz, Crampton, Davenport,* Dun- 

 ham, Emerson, Ewing, Field, Flexner, Gies, 

 Herter, Levene, Levin, Lusk, Meltzer, Men- 

 del,* Morgan, Noguchi, Norris, Oertel, Opie, 

 Richards, Salant, Sweet, Torrey, Wallace, 

 Wolf. 



Members elected. — Joseph Erlanger, E. O. 

 Jordan, Otto Folin. 



ABSTRACTS OF REPORTS OF ORIGINAL 

 INVESTIGATIONS.f 



Contributions to the Study of Sulfur. I. The 

 Metabolism in Brombenzol Poisoning: W. 

 Mackim Marriott and C. G. L. Wolf. 

 Administration of brombenzol to dogs re- 

 sulted in increased elimination of nitrogen 

 and urea. Urea closely followed total nitro- 

 gen. Preformed ammonia was decreased. 

 Creatinin elimination was not appreciably 

 ^fleeted. Total sulfur excretion was not in- 

 creased, but there was almost complete sup- 

 pression of alkali sulfates. Excretion of 

 neutral sulfur, represented for the most part 

 in this case by parabromphenyl-mercapturic 

 acid, was increased 400 per cent. Ethereal 

 sulfate elimination was markedly increased. 

 Total sulfate-sulfur was diminished. Excre- 

 tion of chlorin and phosphorus was practically 

 unaffected. Nitrogen and fat were increased 

 in the feces. The drug caused ulceration of 

 the stomach and intestines, and degeneration 

 of the liver and kidneys. 



* Non-resident member. 



t The abstracts presented in this account of the 

 proceedings have been greatly condensed from ab- 

 stracts given to the secretary by the authors 

 themselves. The latter abstracts of the reports 

 may be found in current numbers of American 

 Medicine and Medical News. 



On Experimentally Produced Variations in the 

 Energy of Tumor Growth: Leo Loeb. (Pre- 

 sented by James Ewing.) 

 The author's observations point to the gen- 

 eral conclusion that it is possible to cause an 

 experimental increase or decrease in the en- 

 ergy of tumor growth. Such variations may 

 be brought about by direct stimulating or de- 

 pressing influences on the tumor cells. The 

 stimulation effects may become cumulative. 



Demonstration: Photographs and Plumage- 

 charts of Hybrid Poultry, with Remarlcs: 

 Charles B. Davenport. 

 Dr. Davenport exhibited photographs and 

 plumage-charts of four hybrids between dif- 

 ferent races of poultry, and also of their 

 parents, and remarked on the nature of the 

 inheritance illustrated by each example. 



Experimental Cirrhosis of the Liver: Richard 

 M. Pearce. (Presented by Eugene L. Opie.) 

 Necrotic lesions were produced in the liver 

 of the dog by injections of hemolytic immune 

 sera of high hemagglutinative power. The 

 author's observations have demonstrated that 

 cirrhosis may follow extensive primary de- 

 structive lesions produced in this way (a view 

 not yet fully accepted) and thus support the 

 contention of Kretz that cirrhosis is essen- 

 tially the result of a series of repair processes 

 following repeated injuries of liver paren- 

 chyma. 



Experimental Arteriosclerosis: Richard M. 

 Pearce and E. McD. Stanton. (Presented 

 by J. E. Sweet.) 



Intravenous injections of adrenalin produce 

 in rabbits vascular lesions that are limited 

 to the aorta and that exhibit more or less 

 definite sequence. ' Five to six injections of 

 3 to 25 minims of 1-1,000 solutions every 

 21^48 hours for long periods cause at first 

 histologically important changes in the media. 

 After about 12-15 injections vei-y definite 

 lesions are evident macroscopically. In the 

 ex[3eriments continued for 6-8 weeks, the 

 process becomes very diffuse and small dila- 

 tions of the thinner portions of the aorta as- 

 sume the appearance of aneurisms. At this 

 stage the destruction of the elastic fibers is 



