S YMPTOMA TOLOG Y 



1867 



and cause dense cellular infiltrations, giving rise to fistulse, which 

 may spread and infect a large cutaneous area. 



When the ova reach the liver by the blood-stream, they give rise 

 to fibrous-tissue formation or an abscess. The surface of the liver 

 shows in places a whitish network and also fiat, china-white plaques. 

 On section a marked increase in Glisson's capsule may be seen, the 

 portal vessels lying in circular or shghtly oval areas of connective 

 tissue. Gall-stones may also form round the ova in the gall-bladder. 

 The pancreas and spleen may also be infected, and the vulva and 

 vagina. 



The ova may also reach the lungs, causing a chronic interstitial 

 pneumonia, and, passing through these organs, may enter the heart 

 and the general circulation. 



Fig. 770. — Liver in Intestinal Schistosomiasis. 

 (After Symmers.) 

 Shows the increase of fibrous tissue in the portal canals. 



Symptomatology.— The incubation period seems to be one to three 

 mofiths. The clinical appearances of the disease may be classified 

 into four varieties: — 



1. Slight infections. 



2. Schistosomic dysentery. 



3. Schistosomic tumours. 



4. vSchistosomic fever. 



Slight Injections. — In these cases there are no symptoms, and the 

 disease is discovered by the examination of the faeces by a microscope. 



Schistosomic Dysentery. — The symptoms resemble those of chronic 

 dysentery, consisting of pains in the abdomen and the passage of 

 blood and mucus. The faeces contain the characteristic ova. 

 The attack begins with a hypersecretion of mucus, followed by 

 frequent small motions containing but little faecal matter and much 

 mucus. The result of these frequent motions is to cause prolapse 



