TRYPANOSOMIASIS EXPEDITION TO SENEGAMBIA 
29 
dilated abdominal lymphatics and glands. In none of them were any living trypano- 
somes found. Neither were parasites seen in the dilated vena cava blood which was 
used for injecting animals (successfully). Several c.c. of this diluted blood were 
eentrifuged and examined, with negative results, lor living trypanosomes. 
Cases II and III. — Were both young stallions owned by natives of Bakau. 
The previous history of neither was known. One had been in the village 
for only a year, the other longer. When seen, both were very thin, and one had 
a slight whitish discharge from the eyes. Temperature ot each about 101 0 F. The 
blood ot both horses contained trypanosomes (five to cover) similar in appearance 
to those seen in the remaining cases. The horse which had been in Bakau for only 
a year was said to have commenced to grow thin and weak in August, 1902. No 
oedemata, haemorrhages, discharges, nor marked paresis were seen in either of 
these horses. Mucous membranes were anaemic. Coats were glossy and not staring. 
One ot these horses was living in May, 1903, the other died during December, 1902. 
Case IV. — A stallion, two years old, owned by a native at Bakau. 
History. — The horse was born in Bakau. Illness commenced a month pre- 
vious to our examination on October [5. It had always previously been healthy. 
The mother ot this colt had died ot an illness which presented similar symptoms. 
Clinical Examination. — The animal was very thin, ribs, ilia, and vertebrae 
being very prominent. There were no signs of oedema, nor of discharges from eyes 
or nostrils, of haemorrhages nor of paresis. The coat was glossy and smooth. 
Temperature was 10 1. 6° F. 
A blood examination showed four trypanosomes to a coverslip preparation. 
The emaciation continued, became extreme, and the horse died during the 
night of November 4. There was no autopsy. 
Case V. — A young two-year-old stallion, bought from a native trader at 
Jesshuon (near Bakau), where the horse was born. It was first seen on October 19, 
1902, when it was found to be infected with trvpanosomes, two to a coverslip 
preparation. Temperature at this time was io3'_|_ ,j F. 
Clinical History. — The colt was small, and when first examined was in tairlv 
good condition. Although thin, it was not emaciated, and was constantly ridden 
by its owner. Its appetite was excellent, but, in spite of a good supply of food, it 
became steadily thinner, and commenced to breathe rapidly and in a laboured manner 
even while at rest. Its coat was rough. 
On November 28 it had become considerably thinner, and a scanty whitish 
mucoid discharge was noticed from eyes and nostrils. Emaciation and weakness 
progressed, and on December 1 8 the animal died suddenly. Towards the end of the 
disease, loss of power became a prominent symptom. The hind legs, particularly, 
were noticed to drag and to be weak. 
