156 THE STATE OF THE BLOOD IN INFLAMMATION. 
after it is healed, continue injected for some time. The cause 
of this appears to be, that the anatomising vessels are not yet 
large enough to compensate for loss of those divided in the in- 
jury. The redness may be in part due to a greater quantity of 
blood than usual arriving at the part in consequence of dilata- 
tion of the arteries. “ These two conditions,” adds Mr. Jones, 
“ appear to me, most probably, to be those in which the blood 
and bloodvessels are in chronic inflammation.” 
CHAPTER VIII. — Of the State of the Blood of the Body 
generally in Inflammation. 
In this chapter, the author’s remarks are chiefly founded on, 
or borrowed with acknowledgment from, the chemical researches 
of Lecanu, Andral, and Gavarret, Simon , Popp, Becquerel and 
Rodier, and Zimmerman. With reference to the microscopical 
characters of the blood of the body generally, in inflammation 
of any organ, Mr. Jones appears to admit that the number of 
the white corpuscles is increased. 
CHAPTER IX. — Of the Relation between the State of the Blood 
generally in Inflammation , and the Inflammation itself 
Inflammation, it has been proved, may exist without increase 
in the quantity of fibrin in the blood; it may also exist with- 
out fever; both often supervene on inflammation. The sudden 
increase in the quantity of the fibrin, Mr. Jones regards as the 
cause of the inflammatory fever; when the increase occurs gra- 
dually, as in pregnancy, &c., no fever is present. Mr. Jones 
concludes, that, “ although inflammation may be independent 
of an increase in the quantity of the fibrin, still, when this has 
taken place, the spread and intensity of the inflammation may 
be promoted by it.” 
Our high appreciation of Mr. Jones’s labours is shewn by 
tiie fulness of our analysis of his Essay. Apart, however, from 
its intrinsic merits, we regard this contribution to our know- 
ledge of inflammation as possessing, at the present time, a 
special value, as an antidote to the vague and profitless views 
of those who would have pathologists regard inflammation as a 
mere modification of nutrition, instead of, as it really is, a pro- 
cess sui generis. The essential phenomena of inflammation 
are bona fide anormal, i. e. pathological, as distinguished from 
physiological, and as such must they be studied. 
Medical Times. 
