1 I 6 Bl BE \i OF \MM\l. imh BTB1 . 
and jaws. Respiratory and bnocal muoons membrane dusky red. Pleural cavi- 
:it;iin a large quantity of yellow, oloody fluid, compressing the lunu r - 
than half their normal bulk, in tin- pericardia] sac there is a similar dropeica] 
effusion, part of which haa formed into a soft, yellowish-white cl rident 
plenritis. Lung «lark red, c ami collapsed. Cavities of heart contain 
dark, soft i»l<»<xi clots; slight petechial extravasations on the epicardinm. No 
obvions peritonea] effusion. Liver is dark in color, friable, and deeply blood- 
rged, the lobular boundarieson section being unusually prominent, with <lark- 
red depressed centers. Kidm tion appear congested throughout, capsule 
nonadherent. * * * 
Tli.- stomach ami Intestines often Bhowed abnormal feataree. The 
small intestine (jejunum) frequently Bhowed hyperemia patches <>n 
both the serous ami mucous surfaces, ami the large intestine ami 
stomach in several cases contained considerable quantities of graveL 
The urine was slightly albuminous in two cases. In <>m- Instance, 
where the brain was dissected, iln-re was engorgement of the veins 
and sinuses of the dura mater, which extended "backward into the 
\ essels of the neck." 
The histological examination is described as follow-: 
tdons of the liver tissue reveal an intern -tion of the portal By stem, the 
intralobular capillaries especially being enonuously engorged throughout and the 
livercells compressed and shrunken. There is, however, no marked degeneration, 
and tin- nuclei take the stain in the normal manner. Sections of the kidney 
exhibit a similar capillary engorgement, though less intense. The glomerular 
tufts art- compressed by edematous effusion into their capsules. A degenerative 
— in the cells of the urinary tubules or other marked pathologic changes 
were not demonstrated. In the spleen no distinct pathologic changes are found. 
Lung sections show a marked congestion of the capillary vessels, with edematous 
effusion and occasional hl<> ><1 extravasations, but without cellular proliferation or 
infiltration. There is no evidence of pneumonia or pleru 
7 atment.—As a rule, hogs suffering from the effects of cotton- 
poisoning, if taken from the cotton-?eed ration ami placed on 
rich green pasture, become apparently well in a week. A similar 
result follows when they arc simply deprived of the cotton 
meal of the ration ami given an ordinary grain ration. However, 
Burtis * reports a ease where a pig died during the winter after a 
week's feeding on a straighl corn diet that followed four weeks" feed- 
in a ration of one-fifth cotton-seed meal ami four-fifths corn meal: 
ami Dinwiddie and Duggar bad similar experiences, [n some 
may pass through a Beason of cotton-seed meal feeding ami 
thereafter be indifferent i«> it. Curtis c found thai if a pig lived 
thirty days after the first appearance of trouble it could be regarded 
as immune from the effects of cotton seed, but the experience of others 
>cem^ to contradict this. Dinwiddie d gives two months as the time 
80, M pi Bxpt Sta.; An. Rpts., 1900-01 and 1901-03, Oklahoma 
Brpt. sta. 
. ftpt 1901 03, Oklahoma Bxpt Sta. 
.1. X... 81, T< cae Bxpt Sta ''Bui. No, :»">. Arkansas Bxpt sta. 
1. 
