EESPIE.ATIOK. 
Eespiration is notably deeper, fuller, and more labored from the first than in 
health, as well as increased in frequency; the respiratory acts are labored and 
increased in frequency in proportion to the amount of cyanosis rather than the 
amount of fever, (Gwinn, 1902. ) 
According to IVilson and Chowning (1902a, p. 133; 1903a, pp. 61-65; 1904a, p. 40) 
the respirator}* rate sometimes reaches 60 per minute in the adult, though ordinarily 
it does not run above 36 per minute; like the pulse rate, it is frequently out of all 
proportion to the temperature; it is regular, but usually shallow; it may be labored 
and accompanied by rattling, due to accumulation of mucus in the upper air passages, 
during the last day or two of life; Cheyne-Stokes respiration has not been observed. 
Anderson (1903a, p. 507; 1903c, p. 23) states that the respiratory rate is always 
increased, usually varying from 26 to 40 per minute, in some cases reaching 50 to 60; 
it is regular, but often shallow. He reports hurried respiration for case 37 i Howard’s 
case, p. 12), between 40 and 50 for case 56 (Howard’s case, p. 14), 24 for case 90 
(Brice’s case, p. 16) the second day after onset, 32, two days after onset, increasing 
gradually to 45, for case 97 (Burton’s case), taking his data from Wilson and Chown- 
ing, 1903a. In case 117 (p. 27) the respiration was at first normal, became more rapid 
and labored until a few hours before death, then gradually weaker; 26 and 28 for 
case 118 (p. 28). 
Gates (1903, p. 50) reports in one case that the respiration varied from 30 to 40 
throughout the course of the disease and continued until after all signs of heart action 
had ceased. He (1905, p. 113) reports Cheyne-Stokes respiration during last of second 
week in his case 16. 
In the cases of 1904 respiration became poor (7) in case 2 . the patient 
at times struo-o-lino* for breath on Mav S; Buckle v observed Chevne- 
Stokes respiration in this case. In case 3 it was slightly harsh and 
prolonged anteriorly: it became slightly stertorous on May 12: on 
Mav 13 the lung sounds were verv harsh all over the front and back, 
with large coarse rales: the lungs were rapidly filling with fluid: for 
5 or 10 minutes respiration would be quiet and regular, then very 
rapid (60) for a time: on May 14 the filling of the lungs progTessed 
rapidly, edema well marked: on May 15 there was much noise in 
breathing. In case 5 the respiration suddenly ceased on May 21: 
artificial respiration was resorted to and in 5 minutes the patient 
breathed, was roused, and quite rational: failure of respiration con- 
tinued to occur at intervals, patient apparently forgetting to breathe: 
hypodermics of morphine acted as a respiratory stimulant (Mills). In 
case 7 respiration became harsh, especially on right side. In case 11 
respiration was depressed, falling on June 24 to S (patient receiving- 
large doses of morphine). 
Edema of lungs developed in case 2. 
CoMPAEisoxs. — For Texas fever, see above, page 63. 
In canine piroplasmosis breathing is accelerated, subsec^uently labored, irregular, 
and finally very shallow; in acute cases respirations are 36 to 48 per minute (acceler- 
ated), labored, gasping, and at times, especially in young dogs, accompanied by 
whining sounds; examination of thorax negative. 
