73 
PLEURAL CAVITIES. 
No adhesions were reported in cases 107, 89, 91, and 120. In case 93 the pleura 
was intensely adherent over entire surface of l)oth lungs; the adhesions were very 
thick and fibrous; there was a history of pleurisy several years before. (Wilson and 
Chowning, 1903a, pp. 47, 51, 52, 54; Anderson, 1903c, p. 33.) 
In 190d the pleural cavities of case 11 vrere normal except a fetv 
adhesions between left lung and pericardium. 
CoMPARisoxs. — In canine piroplasmosis, the peritoneal and thoracic cavities may 
contain fluid (Hutcheon); sometimes there is brownish serous exudate in thorax; 
in Lounsbury’s chronic case there were pericarditis and pyothorax. 
LUXGS. 
See also Gangrene; page 50. 
Wilson and Chowning (1904a, pp. 41, 42) report hypostatic congestion; all the lungs 
show considerable congestion and swelling of the capillaries; many red blood cells 
containing parasites are present; in most cases many phagocytes are found which 
have taken up infected red cells and pigment granules; in one case there was con- 
siderable broncho-pneumonia; pleura was normal. 
Anderson (1903c, pp. 33, 38) reports that the plurse are normal and do not contain 
excess of fluid; lungs show hypostatic congestion, occasionally pneumonia; in case 
120 there were no consolidated areas, except a few points resembling emboli. 
See also Complications, page 87. 
Upon autopsy, the lung tissue in case 93 was fully crepitant; there was slight hypo- 
static congestion on both sides; in cases 89 and 91 both lungs were normal, except 
hypostasis on both sides; in case 107, the lungs were apparently normal, no pneu- 
monia (Wilson and Chowning, 1903a, pp. 47, 51, 52, 54). Incase 120, the lungs were 
normally infiated, no consolidated areas, except a few points resembling emboli 
(Anderson, 1903c, p. 33). 
During the season of 1904, case 2 died of edema of the lungs; in case 
3 also edema of the lungs developed; in case 8, slight edema of the 
lungs developed; there was edema of the right lung May 29, and on 
May 30 both lungs were edematous. In case 11, on autopst^, the right 
lung was lead color on upper surface, very dark posteriorly; several 
dark spots one-eighth inch in diameter anteriorly, but apparent!}" old; 
entire lung edematous; posterior portion extremely congested, in a 
condition of hypostatic pneumonia, and sinks in water; left lung shows 
same appearance as right, except pneumonic area is less marked and 
less extensive. 
Comparison. — In Texas fever the lungs are, as a rule, healthy; there is, in many 
cases, pulmonary edema, with or without emphysema, noticeable after death; in a 
few instances foci of dark red hepatization were observed in one of the principal 
lobes, which involved one or several lobules. (Smith and Kilborne, 1893, p. 26. ) 
In canine piroplasmosis the lungs are rarely affected (Hutcheon); Nuttall noted 
edema and pinkish frothy fluid in the bronchi and trachea; in France apoplectiform 
foci have been found; in young dogs dying quickly, usually there are acute edema 
and reddish foamy secretion in bronchi and trachea. 
Hypostatic lobar pneumonia herds have been recorded in carceag. 
