63 
PULSE. 
Idaho. — The pulse is more or less accelerated, often greatest at the beginning of 
convalescence; in the onset it is sluggish and lacks force; in fatal cases it is not 
! ' usually greatly quickened; it may be slower than in health (Bowers, 1896, p. 63). 
Collister (1896, p. 63) says that it does not run very high, not often above 110 in 
i adults. Fairchild ( 1896) reports it as usually slow and full, from 85 to 110. Springer 
I (1896, p. 62) gives it as 100 to 130 in ordinary cases. Maxey (1899, p. 435) reports 
! it from 80 to 120 per minute, at first full and bounding, later becoming soft, but not 
I irregular. 
J Montana. — At first the pulse is full and strong, it gradually gains in rapidity and 
loses in strength and volume (Gwinn, 1902). According to McCullough (1902, p. 
226) the pulse varies from 80 to 120 in typical cases, and lacks volume and regularity 
as the disease advances. Wilson and Chowning (1902a, p. 133; 1903a, p. 63; 1904a, 
p. 38) say that at the onset the pulse is usually full and strong, but gradually 
. becomes more and more rapid while it loses in volume and strength, very much as 
in diphtheria; in fatal cases in adults it may reach 150 per minute some days before 
death; the rapidity of the pulse is sometimes out of all proportion to the tempera- 
ture, as may be also the respiration. 
' Anderson (1903a, p. 507; 1903c, p. 22) reports that the pulse appears out of all 
! proportion to the temperature, usually running from 110 to 140. A pulse of 120 is 
not unusual with a temperature of 102°; it is ratlier thready, though sometimes full 
; and strong, occasionally dicrotic in the first week (1903c, p. 22) . In case 120 (1903a, 
p. 29) the circulation was feeble on compressed areas and extremities. 
; Gates (1903c, pp. 48, 49) reports 186 as the highest pulse rate observed; one case 
; at the end of the first week was almost pulseless; he reports (1905, p. 112) slow pulse 
i and repeated chills as special features of his case 14. 
j Comparison. — In Texas fever the pulse and respiration rise with the fever. * * * 
As the fever subsides and recovery begins the great weakness of the animal still 
keeps the pulse very high for a time, especially when the animal is moved about or 
I excited in any way. The respirations, on the other hand, are apt to fall below the 
I normal in this same period. When death approaches the heart beats increase in 
number as they grow feebler, and the respirations fall with the body temperature 
below the normal. (Smith and Kilborne, 1893, p. 18.) 
I ln canine piroplasmosis the pulse is weak and rapid; in acute cases it beats 120 to 
160 a minute, is threadv, and often intermittent. 
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' HEMORRHAGE. 
See also Epistaxis, page 52. 
I There is a marked tendency to hemorrhage — nose, throat, lungs, and bowels, each 
have been known to be affected, and the blood loses power of coagulation. (McCul- 
lough, 1902, p. 226.) 
BLOOD. 
Referring to their table, Wilson and Chowning (1904a, pp. 39, 40) conclude that the 
blood shows a marked reduction of red blood cells and hemoglobin, with a slight 
i] , increase of leukocytes at times; the reduction of red cells is jiarticularly marked just 
! before death in fatal cases, and in recovering cases just before convalescence. Prepa- 
rations taken from organs at autopsy, as well as those from the living patient, show a 
■j marked poikilocytosis and anemia. (Wilson and Chowning, 1903a, p. 67.) 
I ' None of the cases 1 to 11 (1901) were in a condition which would 
strike the observer as being anemic. A blood count might have shown 
j 7 some tendency to anemic condition, l)ut from the general appearance 
of the patients such condition was not evident, 
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