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EESPIRATION. 
Eespiration is notably deeper, fuller, and more labored from the first than in i 
health, as well as increased in frequency; the respu’atory acts are labored and ' 
increased in frequency in proportion to the amount of cyanosis rather than the 
amount of fever. (Gwinn, 1902.) I 
According to Wilson and Chowning (1902a, p. 133; 1903a, pp. 61-65; 1904a, p. 40) 
the respiratory rate sometimes reaches 60 per minute in the adult, though ordinarily 
it does not run above 36 per minute; like the pulse rate, it is frequently out of all 
proportion to the temperature; it is regular, but usually shallow; it may be labored 
and accompanied by rattling, due to accumulation of mucus in the upper air passages, ■ 
during the last day or two of life; Cheyne-Stokes respiration has not been observed. 
Anderson (1903a, p. 507; 1903c, p. 23) states that the respiratory rate is always j 
increased, usually varying from 26 to 40 per minute, in some cases reaching 50 to 60; 
it is regular, but often shallow. He reports hurried respiration for case 37 (Howard’s 
case, p. 12), between 40 and 50 for case 56 (Howard’s case, p. 14), 24 for case 90 ■ 
(Brice’s case, p. 16) the second day after onset, 32, two days after onset, increasing 
gradually to 45, for case 97 (Burton’s case), taking his data from Wilson and Chown- 
ing, 1903a. In case 117 (p. 27) the respiration was at first normal, became more rapid 
and labored until a few hours before death, then gradually weaker; 26 and 28 for 
case 118 (p. 28). 
Gates (1903, p. 50) reports in one case that the respiration varied from 30 to 40 , 
throughout the course of the disease and continued until after all signs of heart action * 
had ceased. He (1905, p. 113) reports Cheyne-Stokes respiration during last of second ; 
week in his case 16. , 
In the cases of 1904 respiration became poor (7) in case 2, the patient I 
at times struggling for breath on May 8; Buckley observed Cheyne- 
Stokes respiration in this case. In case 3 it was slightly harsh and 
prolonged anteriorhy it became slightly stertorous on May 12; on 
May 13 the lung sounds were very harsh all over the front and back, 
with large coarse rales; the lungs were rapidly tilling with fluid; for 
5 or 10 minutes respiration would be quiet and regular, then very 
rapid (60) for a time; on May 14: the Ailing of the lungs progressed 
rapidly, edema well marked; on May 15 there was much noise in i 
breathing. In case 5 the respiration suddenly ceased on May 21; 
artificial respiration was resorted to and in 5 minutes the patient 
breathed, was roused, and quite rational; failure of respiration con- 
tinued to occur at intervals, patient apparently forgetting to breathe; 
hypodermics of morphine acted as a respiratory stimulant (Mills). In 
case 7 respiration became harsh, especially on right side. In case 11 
respiration was depressed, falling on June 24 to 8 (patient receiving 
large doses of morphine). 
Edema of lungs developed in case 2. 
CoMPAEisoxs. — For Texas fever, see above, page 63. 
In canine piroplasmosis breathing is accelerated, subsequently labored, irregular, 
and finally very shallow; in acute cases respirations are 36 to 48 per minute (acceler- 
ated), labored, gasping, and at times, especially in young dogs, accompanied by 
whining sounds; examination of thorax negative. 
