IXTERCOMMtiNlC'ABILlTY OF TXJBER'Ct-LOSIS. 
nil 
deaths were due to the maternal cachexia. The sow gradually grew 
sour-tempered, had a husky dry cough, and, when bustled afeiit the 
stye, respiratory distress was very marked. An attempt was made to 
fatten her ; and, although she did scant justice to the process, there 
was improvement sufficient to effect her sale at auction. She was 
secured by a local butcher, and I was present when she was slaughtered. 
The intestinal and mesenteric lymphatics were principally affected with 
characteristic tubercular lesions, but the lungs were also extensively 
diseased — the pleural membrane, both costal diaphragmatic and pulmo- 
nary, being, in the words of the slaughterman, u one mass of grapes.” 
While being able to observe minutely the manifestation of disease 
in the young sow pig just referred to, I had the privilege of access at 
will to the four pigs of the same litter located at the district work- 
house piggeries. The condition and progress of the whole of them 
was almost exactly parallel with that of the one L have just described. 
They had intermittent diarrhoea, did not grow to any size, and an 
attempt to get them ready for the Easter market as young porkers 
totally failed. They were kept on till the end of the summer; and 
every means to improve their growth and condition failing, they were 
sold to a u slink” butcher, who, after keeping them for a little while 
and failing to freshen them up, had them slaughtered and their 
carcasses disposed of in away known only to himself. In confidential 
conversation with this man some time afterwards he stated that while 
he kept them they had chronic diarrhoea and a wheezy cough, and he 
described to me the appearance of the internal organs after slaughter. 
He described the lungs as having cheesy masses in their substance, 
the ribs and skirting (diaphragm) as being “graped,” and the net of 
the bowels or mesentery as being studded with grey nodules and 
tumours of varying size — appearances which to the naked eye are 
characteristic of tuberculosis, and which were identical with those of 
the first pig I described. 
If anything further were needed to confirm or intensify the con- 
viction that these pigs contracted the disease by imbibing the milk of 
the tuberculous cow, I would mention that they were of the sow s 
second litter. Her first litter was healthy, throve, and grew well and 
strong, and none of the pigs from it that were retained on t tie farm 
ever showed the least sign of being diseased. Lhe sow herself was 
healthy, well grown, and lusty. She reared third and fourth litters 
by the same boar, w r hich were uniformly free from un thrift i ness or 
taint of disease ; and when she was fattened and slaughtered all the 
organs and tissues were found to be quite normal. 
At the time the young pigs were being fed on the tuberculous 
milk seven heifer calves also received a portion of it along with milk 
from other cows twice daily. Two of these were sold and lost sight 
of, but the remaining five all died of tuberculosis during from three 
to twelve months afterwards, post-mortem examination revealing 
extensive intestinal and pulmonary tubercular lesions. In this case, 
too, the negative evidence afforded by older and younger calves points 
directly to the milk of this particular cow as being the only means of 
infection, for older calves which were being fed on skimmed milk from 
other cow’s at the time these seven were having the infected milk, and 
younger calves born after the slaughter of this diseased cow grew up 
healthy and strong, and showed no signs whatever of tuberculosis. 
