100 
MASTICATORY PARALYSIS. 
and had afterwards slept for a long time. When it awoke next day, its mistress 
remarked that its tongue was protruding, and that it was unable to close its 
mouth. The animal appeared low-spirited ; the under jaw hung flaccid ; and 
when pressed into position again fell. The tongue was dry and hung out; 
food could not be taken, though pieces of meat placed in the mouth were 
swallowed with great relish. During the next few days the dog appeared quiet, 
somewhat apathetic, and lay continuously in a corner of his kennel. Five days 
later, the disease had already so far yielded to expectant treatment that the 
animal could again take nourishment regularly. 
A similar case, which, however, appeared incurable, occurred in Moller’s 
practice in 1884. . A nine months old dog, used for drawing a barrow, showed 
complete paralysis of the nerve, could not withdraw the tongue, the mouth 
remained continually open, and in taking fluid nourishment or water, the head 
was thrust into the fluid as high as the eyes. The muscles of mastication, 
and particularly the temporal muscles, showed marked atrophy. The tongue 
and buccal membrane were insensitive to injuries ; twitchings occurred in 
the muscles of the rump, like those often seen following distemper ; no 
improvement appeared after lengthened observation and faradisation. As the 
owner objected to have the animal killed, a post-mortem unfortunately could 
not be made. 
Doll and Lydtin saw cases of one-sided trigeminal paralysis in the horse. 
Roll states having found all three branches paralysed; the skin and mucous 
membrane of the affected half of the head and the cornea being insensitive. 
Salivation, difficulty in chewing, and accumulation of food between the cheeks 
and teeth existed. The mucous membrane of the nose, mouth, and conjunctiva 
was hyperaemic. Owing to inability to close the lids and protect ’the eye, 
corneal ulcers formed. Post-mortem discovered fatty degeneration of the 
roots of the nerve and meningitis at the base of the brain. Lydtin observed 
the disease in an old horse. Marked atrophy of the masseter and temporalis 
muscles of the right side had been developing for ten years, swallowing was 
difficult, salivation existed, the molars showed partial shear-mouth. & The 
animal was regarded as incurable, and killed. Post-mortem showed the 
masseter, temporalis, and pterygoid muscles completely atrophied; their 
weight being only one-eighth of those of the healthy side. At the base of the 
petious temporal bone, and just over Gassers ganglion, lay a fibro-sarcoma 
as large as the cerebellum, which must be regarded as having caused the 
paralysis. Frohner considered one case occurring in his experience to have 
been caused by catching cold. 
Cadeac saw trigeminal paralysis in a dog. Recovery occurred in a 
week. Schmidt recorded a case where a sporting dog showed sudden 
paralysis of the lower jaw and marked convergence of the optical axes. 
The animal recovered in one week. 
The symptoms consist of salivation, protrusion of the tongue (which 
is often dry on the surface), and inability to take nourishment, to masti¬ 
cate, or to close the mouth completely. These symptoms, and the fact 
that the mouth can easily be closed by pressing on the lower jaw, 
distinguish this condition from dislocation of the lower jaw, or from 
foreign bodies lodged between the molars. Masticatory paralysis, as 
stated, is a constant symptom of rabies in the dog. 
Prognosis must be based on general principles. Where the paralysis 
has only existed a short time, and is incomplete, some hope may be 
