CONNECTED WITH THE BEAT OE THM MAMHAFJAN HEART. 
171 
Experiment 2 .—Kittens heart. April 1 st, 1888. 
Time after 
fleatli. 
.V to Hg. 
A to H so.,. 
min. 
var. 
Spontaneous beat. 
Heart excised— 
i) 
SN 
Spontaneous beat. 
sN 
., after injury of apex. 
N 
,, ,, ,, and of base. 
1 
..N 
Experiment 3.— Cat's heart. April 21 st, 1888. Death by decapitation. Five 
minutes after death the apex of the heart, consisting of the left ventricle, rvas pulse¬ 
less and in firm rigor ; the l^ase of the heart, consisting of the right venti’icle, ^va.s 
at the same time regularly contracting about 30 per minute ; the auricles were also 
regularly contracting at a rate of 120 per minute; this condition was observed till 
20 minutes post mortem. 
Apex to Hg. 
Liver to Hm 
O 
Neck to Hg. 
Neck to Hg. 
Apex to Hg. 
Base to H^SO^. 
Base to HgSO^. 
Base to HoSO^^. 
Auricle to H 2 SO 4 ,. 
Neck to H 2 SO 4 . 
A^ariation N. 
N. 
N. 
? 5 
n. 
0. 
Remarks .—The condition of the ventricles was such that the right ventricle formed 
a loose pulsating pouch, connected with the upper two-thirds of the firmly contracted 
left ventricle. The contractions of the right ventricle were regular but small, and 
visible only at the basal part; the electrometer indicated negativity of the contracting 
portion. If, with one leading-off electrode applied to an indifferent part, the other 
leading-off electrode was shifted to a distance from the actually contracting portion, 
the excursion was quickly lessened and lost; if it was shifted to the auricle, the 
N variation of a ventricular rhythm gave way to the much more frequent variation 
n of auricular rhythm. 
Nothing can be clearer than these effects of injury at base and apex respectively. 
The diphasic variation SN (viz., apex negativuty followed by base negativity) is, 
in Consequence of injury of the apex, converted into the monophasic variation N 
(unbalanced negativity of base). iAfter a time the diphasic variation SN re-appears, 
and now it is converted into the monophasic variation S (unbalanced negativity of 
apex) in consequence of injury of the apex. These facts, illustrated in fig. 1 , are 
precisely similar to those observed by Burdon Sanderson and Page upon the 
ventricle of the Frog and Tortoise, the only difference being in the nature of the 
normal variation antecedent to injury. 
* ‘ Journal of Physiology,’ vol. 2, p. 418 ; vol. 4, p. 33.5. 
z 2 
