VETERINARY MEDICAL SOCIETY. 
119 
As the disease proceeds, pulse more frequent, small, wiry; 
respiration accelerated ; tongue furred ; full inspiration; difficult 
and painful expiration; patches of sweat; pulse more indistinct, 
restlessness, delirium, death. 
Symptoms of first stage continue from three to seven days, 
when the disease terminates fatally, or symptoms of the second 
stage commence. The contractions of the abdominal muscles 
diminish; belly drops; full inspiration; less painful expiration; 
convulsive twitching, less violent and less frequent; pulse dis¬ 
tinct and soft, but quick ; alternations of remission and exacer¬ 
bation. In favourable cases, the quickness of pulse gradually 
diminishes; body regains its flexibility; appetite returns; the 
horse lies down; and in three or four weeks may be considered 
well, although unfit for work. 
The post mortem appearances are sometimes injection and 
deepened colour of the costal and diaphragmatic pleurae, with¬ 
out effusion; sometimes the lateral pleurae highly vascular, with 
numerous filaments from the inflamed surface, and patches of 
lymph, with a small quantity of sero-purulent effusion. 
In most cases a considerable collection of fluid, pale, or yellow, 
or reddish, or sanguineous, in all three cavities; or serous on one 
side, and sanguineous on the other; the lateral pleurae thickly 
coated with lymph, and adhering to the pulmonary pleurae ; at 
times, stripes or patches of inflammation; occasionally one lung 
surrounded by a complete adventitious membrane ; the lungs 
generally flaccid, less crepitant, and considerably compressed. 
From contiguity, the pericardium frequently vascular or coated 
with lymph, with effusion in its cavity. 
He had had a few opportunities of inspecting the bodies of 
horses at distant periods after recovery from pleurisy. In some 
no morbid appearance could be detected; in others a broad band 
from one to three inches, with interposed blood-vessels, extended 
between the pulmonary and costal pleurae. 
At an early period of the discussion, some gentlemen ex¬ 
pressed a doubt as to the existence of pure pleurisy. One gentle¬ 
man considered pleurisy as the consequence and proof of chronic 
inflammation of the parenchymatous substance of the lungs. It 
was replied, that the symptoms which characterised pleurisy were 
altogether different from those of chronic inflammation of the 
substance of the lungs. 
The partial and local inflammation was denied. It was as¬ 
serted, that inflammation of a serous membrane must be equally 
diffused through the whole of it. It was replied that facts were 
stubborn things, and that this confined extent of inflammation 
