W. L. Braddon 
275 
Figs. 13-16. Some larger forms, partly extra-corpuscular. Note in 15 complete “trans¬ 
fixion ” of erythrocyte. 
Figs. 18-21. A single body drawn at intervals of 10 secs., showing rotation on long axis. 
Figs. 22-24. Almost completely detached body drawn at intervals of 10 secs. 
Figs. 25-27, and 28-30. Other single intra-cellular specimens drawn at similar intervals. 
Figs. 31-34. Slender forms (stunted) from blood of partially immune Siamese cattle. 
Figs. 35-51. Various examples from buffaloes. 
Fig. 52. From buffalo during infective stage. Appearance suggesting full-grown body 
completely filling corpuscle, converted into granules at limits of resolvability (the dots 
in the drawing are too large)—infective granules ? 
Fig. 53. Cell full of larger (growing?) comma forms from buffalo during infective stage, 
5th day. 
Fig. 54. Multiple young forms from buffalo, early stage of infection. 
Figs. 55-57. From pig artificially inoculated with virus from buffalo, five days after 
infection. 
Figs. 58-60. Buffalo, fourth day after inoculation. 
Figs. 61-70. Multiple forms, from a single field, in blood of a buffalo-contact, which at 
date of examination appeared healthy (no temperature) but three days after was dead 
of an “algid” (hyperacute) attack of rinderpest. 
Fig. 71. Scalene triangle form, very common. Note only margins of inclusion are 
indicated, membrane between them unstained. 
Figs. 72-77. Free forms, commonly seen in plasma in buffaloes after 4th or 5th day of 
infection ; stained and unstained. 
Figs. 78-109. Red corpuscles with distortions indicating presence of specific inclusions, 
but in which no staining having taken place they are not defined. 
Figs. 78-84. Typical “ squared ” corpuscles. These are often seen within 12 hours and, 
before staining, are pathognomonic as indicating presence of specific inclusions. 
Figs. 79, 80, 83. Normal red corpuscles drawn beside the squared corpuscles for com¬ 
parison. 
Figs. 85-109. Various shapes assumed by extruding specific bodies in buffalo blood. 
These are refractory to stain in the fluid condition, but take on all plasmatic stains 
equally with the haemoglobin in fixed specimens. 
Fig. 110. Form assumed by red corpuscles of goats infected with rinderpest. 
Figs. Ill, 112. Late atrophied forms of intra-corpuscular bodies in buffaloes eight 
months to a year after recovery from rinderpest. 
Figs, a—/ were drawn from actual specimens by Miss Rhodes at the Lister Institute. 
Figs, a — e. Intra-corpuscular bodies occurring in rinderpest. Blood stained, while fluid, 
with methylene blue citrate of potash solution, and afterwards fixed and restained. 
Fig. a. Slightly counterstained with Eosin (colour omitted in plate). Only the edges of 
the inclusions appear to be stained. 
Figs, h — e. After treatment with Orange G. and Anilin blue. A definite expanse of 
membrane between the edges is shown, (In the original figure the inclusions appear 
brownish.) 
Fig. /. Blood from a case resembling rinderpest. Group of corpuscles in a microscopic 
field. Blood fixed in perchloride of mercury after staining, while fluid, with methyl¬ 
ene blue citrate of potash solution. 
