320 
Haemog i eg ar hies 
parations which were taken from the living animal and immediately fixed,” 
and by Schubotz (1913), who observed very numerous free forms of H. pettiti 
(from Crocodilus niloticus) in the blood from peripheral vessels and from 
internal organs (liver and spleen). In fresh preparations sealed with vaseline 
this author kept the free forms living for hours. To this question I cannot 
give a definite answer based on my own material, but judging from pictures 
like Figs. 10 and 11, and from the complete absence of these forms within 
the blood corpuscles, it is easier to agree with Seidelin. The phenomena 
observed by Stebbins and Dobell give further confirmation of it. 
Type 2 is represented by intracorpuscular forms of different size; one end 
of their body is much thickened, the other terminates in a short point, the 
body being bent over in different degrees (Figs, 12, 13, 14). The nucleus is of 
the usual vesicular structure, and the protoplasm is granulated, the granula¬ 
tion being coarser than in the preceding form. Sometimes the protoplasm 
contains dark staining lumps, probably of the so-called volutin (Figs. 13, 14). 
The blood corpuscle infected by this form is hypertrophied (Fig. 13) and its 
nucleus undergoes degeneration (Fig. 12). 
The most numerous are forms belonging to 
Type 3. All of these are intracorpuscular, the body is of an irregular bean¬ 
shaped form, measuring 18-8 /x x 7-8p,. The nucleus of the parasite is disposed 
nearer to one of the ends of the body, the protoplasm is granular, and probably 
sometimes contains volutin granules (Fig. 15). This form is, moreover, charac¬ 
terized by the constant presence of a kind of dark cap situated on one end 
of the body (Figs. 15, 16). This formation evidently presents the frequently 
described accumulation of excretions of the parasite (Dutton, Todd, and 
Tobey, 1907; Stevenson, 1911; MacFie, 1914, and others). 
The influence of this form of the parasite on the cell harbouring it is 
expressed in a general shrinkage of the erythrocyte, the body of which is 
ultimately reduced to the condition of a thin membrane enclosing the parasite. 
At the same time the nucleus of the erythrocyte degenerates and often under¬ 
goes fragmentation, breaking up into two parts (Fig. 16), as has been already 
frequently described by other authors (Stebbins, 1905; Plimmer, 1912; Conor, 
1912; Reichenow, 1912; Shortt, 1917). This form resembles the haemo- 
gregarine described by MacFie (1914) from African toads, and, in some 
respects, that described by Shortt from Bufo melanostictus . 
In all the types described above, the parasite is surrounded by a distinct 
light rim. 
To type 4 belong parasites whose body is club-shaped, or of irregular form, 
from 19 /JL to 34/x long, characterized by a large nucleus of round-oval form 
and vesicular structure (Figs. 17, 18). All forms of this type were encountered 
free in the plasma and only in one case a parasite was found in the act of 
leaving the host cell, in which the remains of the abandoned capsule are 
visible (Fig. 17). Similar forms were described by Dutton, Todd, and Tobey 
(1907), Flu (1910), and others. 
