26 
APOPLEXY IN A HORSE. 
The animal died about two hours afterwards. 
At the desire of Mr. Greenaway 1 made a post-mortem 
examination. On opening the abdomen I found a large 
quantity of bloody liquid existing within the cavity, and on 
examining the stomach I found a rupture in it to the 
extent of six or eight inches, extending across the great 
curvature. The walls of the viscus were pallid, and of 
so weak a texture that they were torn as easily as a piece of 
blotting-paper. The stomach itself w T as nevertheless crammed 
with dry food, some portions of which had escaped through 
the opening. 
The mare was always a hearty feeder, and in my opinion, 
from her constantly overloading the stomach previous to 
work, it so weakened this organ that a rupture w r as the ulti¬ 
mate result. 
CASE OF APOPLEXY IN A HOUSE. 
By the Same. 
I was sent for on the 3d of October to see a horse 
belonging to a farmer, which I was informed had “ staggers.” 
On my arrival I found a four-year-old cart-colt, labouring 
under congestion of the brain, and almost completely 
paralysed. He was perfectly helpless, and when moved 
fell down. The pulse was about 50 in the minute, but 
scarcely to be felt; the conjunctival membrane was much 
inflamed ; the iris not contracting at all on a light being 
held near the eye; the breathing stertorous, and the bowels 
constipated. 
I immediately bled the animal, taking away nearly seven 
quarts of blood (the pulse rising directly after), and gave 
Aloes Bbd., $ v j ; 
Hjd. Clilor., $j; > 
Sp. Eth. Nit., 5j, in kaustus. 
Applied sinapisms containing ammonia to the head, and 
ordered clysters to be frequently thrown up. 
October 4th.—Partial consciousness had returned, but 
the loss of power in the extremities still existed, and the 
constipation of the bowels. I gave 
Aloes Bbd., ; 
Hyd. Chlor., 5j ; 
Sp. Etk. Nit., §j, in kaustus. 
