BEE V. HAYWARD. 
322 
pulse ; loss of appetite ; maintaining standing posture ; fore legs 
abducted, &c. No nasal discharge ; which I mention because, on 
post-mortem examination, I found abscesses in the lungs, which 
many will, no doubt, admit to be rather singular. After careful 
examination, I found him to be labouring under a severe attack 
of acute pneumonia. My prognosis, however, as I stated to 
the carter at the time, was more unfavourable than usual, on ac¬ 
count of the perceptible dead and grating sounds I found upon aus¬ 
cultation ; giving it as my opinion at once, that, in consequence 
of my patient exhibiting previous disease, and now with this 
acute attack upon him, he was beyond the pale of recovery, 
although he might probably linger a week or ten days. 
I, however, lost no time in treating my patient according, I be¬ 
lieve, to the acknowledged principles of practice, viz. copious 
venesection; counter-irritation by means of blisters; the ad¬ 
ministration of sedative, febrifuge, and slight aperient medicine: 
which treatment I continued daily and nightly, varying it slightly 
owing to alteration in the symptoms, until the animal’s death, 
which occurred on the 20th day following that of attack, when, 
by means of a post-mortem examination, the undermentioned 
state was apparent. 
The abdominal viscera had certainly undergone the change 
that is usual to find, after a week’s illness, depending upon acute 
inflammatory attack of an organ that has to perform vital func¬ 
tions, for the liver, spleen and kidnies were in a state of ramol- 
lissement. On exposing the thoracic cavity, I found the pleura- 
pulmonalis adherent to the pleura-costalis in five separate 
places: there were three on one lung, and two on the other, by 
means of real organized growths, which were not to be detached 
by hand, but required for division the aid of the scalpel, 
which having accomplished, I removed the lungs: hydro- 
thorax was not present. On further examination, the pleura 
pulmonalis displayed elevations on its surface, arising from 
the presence of abscesses and tubercles in various stages of 
development. The parenchymatous structure of the lungs was 
highly inflamed. I have now to add, that, even with the presence 
of acute pneumonia, the tubercles did not appear in an inflam¬ 
matory state, but were of an indolent nature; and the pleurae, 
although there were adhesions consisting of regular organized 
growths, and elevations of the pleura pulmonalis by means of 
abscesses and tubercles, being directly underneath in different 
degrees of formation. There was not the slightest appearance 
of inflammation of that membrane present at death, no thoracic 
serous effusion. Taking all these circumstances into considera¬ 
tion, coupled with, I believe, the admitted fact, that the paren¬ 
chymatous tissue of the lungs is lowly organized, and con- 
