372 
T. F. WINCHESTER. 
vigor of the animal, stiffness in movement of the hind quar 
ters, difficulty and pain in micturition, arching of the loins 
infiltration and intermittent lameness of one or both hind 
limbs, cramps, and signs of paraplegia; but these indications! 
are not sufficiently characteristic to afford a sure diagnosis, 
though they may arouse suspicions, which will sometimes be 
confirmed by a rectal exploration. 
Attacks of colic are the most frequent signs of vermin¬ 
ous aneurism, and are the consequence of embolism in the 
branches of the diseased vessels, but neither are these symp¬ 
toms characteristic. Sometimes the colic is sudden and: 
acute, and disappears in a short time, to reappear after a va¬ 
riable interval; it depends upon local obstruction, which is 
soon compensated for by neighboring anastomoses and is! 
usually ascribed to indigestion, as there is no appreciable 
cause. In other cases the colics are sub-acute and a little 
painful, and are due to sudden paralysis of a portion of the 
intestine. Death soon follows, or the disease runs a chronic 
course, and is characterized by difficult digestion, constipa. 
tion alternating with diarrhoea, slight colic, some fever, and 
a capricious appetite. It is a kind of intestinal catarrh that 
may continue for days or even weeks, and terminates either 
in recovery or, which is more frequent, by marasmus, cache- 
kia and death. 
Lastly, in some cases, the embolic obstruction of the small 
arteries of the intestine, when often repeated, ends in hemor¬ 
rhagic enteritis, to which the animal succumbs in several 
days or weeks. Friedberger and Frohner attribute to this 
state the following symptoms : Diminution of appetite or 
complete inappetence, increased thirsts, and rare defecation; 
the fcecal pellets are small and dry at first, then become soft, 
pasty, and, later, sanguinolent and foetid ; the urine is acid, 
and rich in phosphates ; the fever is intense and persistent, 
and the pulse small and quick ; the general debility increases, 
the animal becomes emaciated, and the abdomen retracted, 
and now and again there is coma. Frequently, after feeding, 
there is general aggravation of the symptoms and colic. 
Death is often ushered in by febrile paroxysms, muscular 
