266 
GEORGE DITEWIG. 
the greater portion of the colic artery. The great mesenteric 
and many of its branches exhibited similar lesions. The caeliac 
axis and branches did not escape, the hepatic at the posterior 
fissure of the liver harboring many of the parasites. 
Along the course of the colic arteries and lesser curva¬ 
ture of the great colon was a chain of tumors varying in 
size from a pea to a walnut. Cutting showed them to be 
abscesses containing a creamy pus in which floated usually J 
one strongylus. Scarcely a blood-vessel of the digestive tract : 
escaped their ravages. Aneurisms of any marked size were ; 
absent. 
Here and there, in the worst affected arteries, were small 
adherent thrombi. Near the pelvic flexure of the great colon, 
between the layers of the mezo-colon, was a large blood- 
clot, perhaps two pounds in weight. This haemorrhage was 
probably due to perforation of an arterial wall by one of the 
parasites, or rupture of one of the small aneurisms of the 
colic artery. No matter where cut, in the arteries could be 
seen the pale pink forms of the undeveloped armatus para¬ 
sites. They numbered throughout, stating it mildly, hun¬ 
dreds. 
This was the only autopsy made, but the history, s.ymp- j 
toms, course and termination being so markedly similar in 
all the fatal cases leads me to conclude that all would have i 
presented similar lesions had the examination been made. 
In closing, will venture some conclusions: 
First, that death was the result of anaemia, marasmus, j 
prostration and collapse. 
That the progressive anaemia was due to the continual in¬ 
terference with the nutritive functions of absorption and as¬ 
similation. 
That the diarrhoea was caused by the disturbed intestinal : 
circulation and irritated intestinal mucous membrane. 
That the weakness and marasmus followed as natural re¬ 
sults of anaemia and diarrhoea. 
That the causes continuing in operation, the effects not 
only continued but became so marked that the vital forces 
gave way, and death followed. 
