come more viscid, cling to the walls of the vessels, and that 
very strange phenomena of their passage through the unrup¬ 
tured parietes is observed. 
Leucocytes migrate from both small veins and capillaries 
while there is diapedesis of haemocytes from the latter only. 
They form 3. thick covering around the minute blood-vessels in 
the inflamed part. 
Comparative stasis has now set and there is little circulation 
through a badly affected part for several days. 
Results. —The results of inflammation are the following: 
Resolution, effusion, suppuration, induration, adhesion and gan¬ 
grene. 
Resolution is seen where the cause is impotent or when the 
whole process is cut short by timely and vigorous treatment, 
leaving the organ in a normal state, with no signs of the prior 
pathological condition. 
Effusion is the outpouring of a large amount of serum into 
the neighboring cellular tissue or some adjoining cavity as seen 
in hydrothorax from pleuritis, ascites from peritonitis, etc. 
Suppuration ensues when the cells of the inflamed tissue are 
changed into those of pus by the presence of some one of 
several different pyogenic microbes. 
Pus commonly springs from areolar tissue or from mucous 
surfaces. 
Induration usually arises from protracted irritation or from 
inflammation of a low, chronic type, especially when located in 
tendinous, ligamentous or dermal structures. 
Adhesion occurs principally where two inflamed serous sur¬ 
faces are held in juxtaposition finally coalescing. Inflammation 
of serous parts tends to adhesion of mucus, to suppuration. 
Gangrene, the death of a large part, is due to incompetency 
or total suspension of circulation. 
Symptomatology. —Among the symptoms of inflamma¬ 
tion are pain, heat, redness, swelling and tension. 
Pain springs from injury to, or pressure of the exudates on, 
the local nerves; in some cases, accordingly, free incision of the 
