3SS 
EDITORIAL. 
manifestation of an infection, first by the lymphatics and later by 
the circulation, are already old. 
7. In young children and young animals, specially sensitive, 
lymphatic and bloody bacillar infection assumes sometimes the 
ways of a septicaemia, without resulting in all the cases with the 
formation of follicular lesions or of tubercles. This septicaemia 
is frequently curable. However, if the tuberculous bacilli are 
not eliminated and if they create centers of symbiotic life with 
the lymphatic cells of the various organs, follicular lesions are 
formed and the subject remains tuberculous. 
This tuberculosis can remain indefinitely or during long years 
as latent, recognized only by tuberculine reactions, and the car¬ 
rier subject shows then a manifest resistance to reinfection. But, 
if the follicular lesions are numerous and disseminated in poorly 
protected organs, they progress more or less rapidly and give 
rise either to acute miliary tuberculosis or to an extensive local¬ 
ized infection. 
8. Some organisms are naturally or may be made artificially 
unfit to undergo tuberculous infection. It seems, however, that 
the required immunity resulting from a mild anterior infection 
or obtained from artificial method, is not of long duration. It 
lasts only as long as the organism remains a carrier of bacilli or 
of latent follicular lesions. It disappears shortly after the time 
when the aptitude to reaction with the tuberculine ceases. 
9. When the tuberculous bacilli enters in an organism, nat¬ 
urally or artificially rendered refractory to tuberculous infection, 
it may either remain as a harmless foreign body, while still keep¬ 
ing its vitality and its proper virulency, or it may be eliminated 
with the cellular dejecta of the organism by the natural emuncto- 
ries, such as the liver or intestine, without losing its vitality nor 
its virulency towards other susceptible individuals. 
It is exceptional that it may be integrally resorbed, the ac¬ 
tivity of the cellular products of the leucocytes being powerless 
to dissolve the surrounding membranes that insure its protection. 
10. The virulency, that is, the infecting power of the tuber¬ 
culous bacilli, varies: 
