BIOLOGY. 
Prot. 41 
pseudopodial filaments of Pterocephalus to the endothelial cells of the 
gut ;—inter- not intra-cellular, Siedle(5ki (357). 
Reciprocal relations between growing Gregarines and the host-cells; 
effects on the latter and their reactions. A polycystid Gregarine entirely 
intracellular, even in the adult stage, in the intestinal epithelium of Poly- 
xenus lagurus , Leger & Duboscq (218). 
Coccidiosis:—Hypertrophy of spermatogonial cells containing Caryo- 
tropha mesnilii ; diffusion stream between the nucleus of the host-cell and 
that of the parasite, Siedlecki (358). 
Coccidium faurei, n. sp., causes progressive pernicious anaemia, sooner or 
later ending fatally, in the Sheep, Moussu & Marotel (268).—Lesions 
produced in the liver and bile-ducts of the rabbit by C. oviforme , Tyzzer 
(395).— C. mitrarium , extracellular, and probably derives nutriment from 
the endothelial cells of its host ( Damonia ) by pseudopodia, in a gregarine- 
like manner, Laveran & Mesnil (202).— C. ranarum , n. sp., intra-nuclear 
parasite of the gut of Rana escidenta , Laveran & Mesnil (206). 
Cyclospora caryolytica is an intranuclear parasite of the intestinal 
epithelium of the Mole, producing enormous hypertrophy of the nucleus, 
and atrophy of the host-cells. Rapid endogenous multiplication causes 
pernicious enteritis, which usually ends fatally, Schaudinn (335). 
I so spar a [. Diplospora ] Ueberlciihni: effects on kidneys and other organs 
of Rana escidenta , Laveran & Mesnil (200).—Intra-nuclear habitat of 
/. mesnili; action on nuclei of the intestinal epithelium, Sergent (354). 
Klossiella muris from the renal epithelium (both tubules and Bowman’s 
capsules) of the mouse. The cells of the tubule containing the parasite 
swell up and occlude the lumen, Smith & Johnson (368). 
Legerella testiculi does not ordinarily affect spermatogenesis in Glomeris ; 
only in the rare case of total invasion of the organ, does castration result, 
CufcNOT (75). 
Haemosporidiosis :—[Note. Many of the following papers, referred 
to generally, the Recorder has not seen.] 
Malaria in general:—B raddon (39), Calmette (54), Dionisi (84) and, 
of bats, (85), Ficalbi (115), Guiart (148), Grassi (143), Kunst (179), 
McElroy (252), Plehn (293), Ruge (324), Schoo (342-344), Schuffner 
( 349), v. Schulthess-Rechberg (350), and Trolard (392). 
Malaria and Mosquitoes: Bordi (26), Brumpt (47), d’Espine (104), 
Eysell (105), Galli-Valerio (in Canton Vaud.) (133), Levick (226), 
Meisenheimer (253), Rees (310), Sambon & Low (330), Wright (416), 
and Ziemann (438) ; in Algeria, Billet (21 & 22), Soulie (369); in Mada¬ 
gascar, Laveran (193); in Corsica, de Bastelica (15), Battesti (16) 
[) Anopheles maculipennis = A. claviger Gr. & Fic., the principal propagative 
agent or definitive host], Belval (18), Boyer (38), and Michon (260); 
and in Japan (Jeso Island), Tsuzuki (393). 
Malaria in Algeria of two classes : (1) cycle sestivo-autumnal or primary 
malaria, and (2) cycle hiberno-vernal, or secondary malaria, which Billet 
( 20) thinks are both caused by different cycles of the same parasite, the 
one, the former, chiefly sexual, leading to the formation of crescents, the 
latter, chiefly asexual, or schizogonous, and constituting “recurrence.” 
He doubts the specificity of the sestivo-autumnal (comparable to the per¬ 
nicious) parasite (at all events for Algeria) but believes in the distinction 
between the tertian and quartan ones, which both have an sestivo-autumnal 
cycle—the first to occur in a fresh subject, Billet (20). 
The recurrence of tertian-fever is due to macrogametocytes “casting- 
back” to schizonts and recommencing merogony, Schaudinn (326); same 
explanation for malarial recurrence in general, Grassi (144), contrast 
Billet (20) above. 
Actual penetration of sporozoites of Plasmodium vivax into the blood- 
corpuscles observed, Schaudinn (336). Appearance of spots in the 
