424 
ON INCREMENT OF ANIMAL HEAT. 
ture than the muscles and organs of the trunk of the body. 
I should add that the observations on the relative temperatures 
of the brain and trunk were determined on the pigeon, rabbit, 
and guinea-pig. 
Brain .—After death from increment of animal heat, the 
brain is found, as far as I can say, simply pale. I have 
never been able to find any exudation or congestion, or 
other lesion. It seems to me as if by contraction of vessel 
the organ had been for some time before death deprived of 
its blood, and this view is borne out by the symptom of 
coma which precedes death. This absence of blood may be 
a reason for the lower temperature of the brain immediately 
after death. 
Lungs .—The lungs after death from increment of heat are 
florid and free of congestion, when, previous to death, there 
has been no separation of fibrinous mass in the heart. In 
cases where fibrine has separated on the right side of the 
heart, the lungs are found pale or white, as is so often seen 
in the human subject after death from similar cause. I have 
not found any structural disease of lung. 
Heart .—The heart is found in one of two states, varying 
with the mode of death. In cases where the increment of 
heat is rapid, and the death occurs from tetanus, the heart 
is found empty of blood, and so firmly contracted on itself 
that it feels like a st.one. In cases where the death is from 
separation of fibrine, the heart is often distended with co- 
agulum, but the muscular walls are not flaccid, and they soon 
pass into rigidity. 
Blood .—The blood is found in one of two con(|itions. In 
cases where the fatal increment of heat is very rapidly de¬ 
veloped, the blood, immediately after death, is quite fluid in 
the heart and vessels, but on removal it coagulates very 
firmly, and without any undue delay. In cases where the 
increment of heat is reached slowly, the blood is found with 
the fibrine in part separated, as we have seen in the specimen 
before us. The same facts may be observed in the human 
body suffering from inflammatory fever, and the recognition 
of them in the inferior animal, under knowm conditions, aids 
us greatly in understanding what seems, without such aid, 
to be an insuperable difficulty. There may be death in in¬ 
flammatory fever from rapid increment of heat and extreme 
contraction of vessel before the fibrine has separated, and 
before there is accumulation of water in the blood to favour 
separation of fibrine. There may be death from slower in¬ 
crement, with separation of fibrine as the final and conclusive 
part of the fatal process. 
