698 IRISH CENTRAL VETERINARY MEDICAL ASSOCIATION. 
The horse was off his feed and declining for some time, standing 
in his box with head towards the door or window, and not lying 
down ; with a soft irritable cough, and short careful breathing, but 
not much increased. The pulse full about 50 to 55, while the 
animal knuckled over on one hind leg constantly ; occasionally 
looking at the flanks, without any secretion from the nostrils, and 
the mouth dry and hot. I was somewhat puzzled at first, and 
suspected the liver, but on using the stethoscope I at once con¬ 
cluded the horse was suffering from subacute or chronic inflamma¬ 
tion of the lungs. 
On inquiry I ascertained the horse had been exposed after a long 
drive to a severe shower of rain, and showed nothing more than 
dulness for ten days after, when he seemed to improve, but in three 
weeks again refused his food and became very weak. When I was 
called in I had the sides well blistered, gave a stimulant, and after 
salivated him with calomel, and kept at perfect rest during the 
treatment. He gradually improved, but was always thick winded, 
depending on, probably, some consolidation of the lungs. 
Many horses have a continuous soft cough, which differs from 
both that of broken wind or the regular “ chronic,” which decrease 
at one time, and on exposure to any wet or chill comes on again ; 
is often dependent on some subacute inflammatory action going on 
in a part of the lung-tissue. They are generally light-made, delicate 
feeders, which perhaps account for their not becoming broken 
winded. Post-mortem examination may disclose small abscesses 
formed, while it may induce the sequel, which is perhaps the most 
common, namely,— 
Phthisis or tubercular disease^ which I may say is rare in the 
horse in comparison to man. As an idiopathic affection it is even 
less frequent, but there is no doubt but that true tubercles are found 
in the lungs of our equine patients. It is unnecessary for me to enter 
into the consideration of the various kind of tubercles, but allude to 
those found in the horse, namely, the miliary, which Lacunie con¬ 
siders the true kind. The production is not only varied, but 
difficult to account for. Hereditary predisposition is not often the 
cause in horses, although it is quite possible it may be conveyed in 
this way; but I am inclined to consider that it is oftenest the result 
or sequel of some inflammatory attack, strangles more particularly. 
In young horses getting the disease on grass, and remaining perhaps 
for days without even shelter or any attendance, the symptoms do 
not run their ordinary course, and particularly if the horse is 
weak. The glandular swellings of the parotid or submaxillary 
glands do not come to anything more than a little induration and 
tulness, and they permanently remain so. There is no discharge 
from the nostrils, perhaps a watery moisture, but no signs of the 
healthy yellow discharge we have in true strangles. There is a 
slight cough, which also continues, but without any marked altera¬ 
tion in the breathing. Great emaciation is perhaps the most 
marked symptom, the muscles of the thighs and shoulders waste, 
and the old coat of hair remains on with the skin fastened to the 
