644 IDENTITY OF PLEURO-PNEUMONIA AND RUBEOLA. 
active congestion, or sthenic inflammation, or even from the 
hypostatic inflammation in adynamic fever, the hepatization 
is more uniform, being deep-coloured and dense at the lower 
portions, lessening as it ascends to the upper. In rubeola and 
in pleuro-pneumonia, it is quite different. The congestion, or 
condensation, whether it be from inflammatory action proper, 
or merely congestion per se , is patchy. A section of a lung, or 
lobe, shows this singularity, and partly gives rise to that 
marbled or mottled appearance so well known to those who 
have inspected them. It was not inaptly likened to a piece of 
brawn by one observer. To this aspect the interlobular 
areolar tissues, loaded with fibrin, and so rendered thicker, 
and in colour more yellowish and distinct from the red, or 
brown, or pink vesicular portions; contribute. As there may 
be co-existing all degrees of congestion, of inflammation, and 
of hepatization, so are there all shades of resulting colour, 
from reddish yellow to deep brown. But these colours never 
merge into one another; they are all bounded by the bands 
or septa alluded to. The grand thing to note in this parallel 
is the isolated portions of the lungs thus separately congested 
in both diseases, evidently from the same form of morbid 
action going on during the progress of the disease. The exu¬ 
dation exudes in the same manner as in ordinary cases of in¬ 
flammation. There are the processes of resorption of serum, 
and organization of fibrin into false membranes, causing 
adhesions between the naturally free surfaces of the pleura. 
It is often correctly stated that adhesions rarely form in the 
exudation upon the pleura in pleuro-pneumonia, and in this 
character also the complaint agrees with rubeola. For this 
reason loose flocculi of lymph float in the fluid of the exuda¬ 
tion. As other terminations or results of inflammation there 
are gangrene and abscesses, according to the degree of inten¬ 
sity, of which pathologists do not require particular descrip¬ 
tion. When an affected lung recovers tone, and is restored 
to healthy function, it is accomplished by the same repara¬ 
tive actions as are known to take place in the human body. 
In cattle, as in man, it often happens that there is neither 
death nor absolute recovery. The lungs remain permanently 
impaired; hectic supervenes, and long after the first attack 
may have been forgotten, the animal sinks under the inroads 
of consumption. In such cases it is occasionally spoken of 
by those who are observant and skilful, yet not pathologists, 
as a recurrence of the original disease lasting through many 
months—as a re-wakening up of the symptoms after they had 
been lying for a time dormant. But it would be just as cor¬ 
rect to speak of a man’s dying of rubeola, who sinks from 
phthisis, induced in a lung weakened by the sequelae of a 
