66 
LIVERPOOL VETERINARY MEDICAL ASSOCIATION. 
objection in that, as the fluid would gravitate to the most dependent 
part. 
Mr. Mam said, in reference to the state of the lung mentioned by 
Mr. Hill as being an important factor in this case, that hepatisation of 
the lung could take place within a period of four days prior to death. 
Mr. Storrar , in reply, said the important point for consideration was, 
Did the cause of death commence at the flanks or at the heart ? 
There was no indication of derangement of the heart’s action when 
the swelling first appeared in the flanks; besides, there was no filling of 
the extremities, effusion into the chest or between the fore legs, as we 
generally find associated with heart disease, or in cedematous swellings 
arising from debility. 
There was no indication either of anything being wrong with the 
chest until after we had been treating her for several days. The hot 
fomentations in his (Mr. Storrar’s) opinion relaxed the tissues more and 
more, and consequently increased the effusion, which led to internal 
congestion. There was tenderness of the throat observed the first day, 
followed by disease of the chest the second day. 
There is no doubt that her violent action immediately previous to 
death greatly aggravated the lacerated condition of the abdominal 
.muscles, but, taking all the symptoms into consideration and the order 
of their development, he could not help arriving at the conclusion that 
the cause of death commenced by rupture of the abdominal muscles, and 
that the clots in the heart did not exist for any length of time prior to 
death. 
Mr. Tom Taylor exhibited a beautiful specimen of a brain taken from a 
horse which had an acute attack of apoplexy. 
The history of the case is briefly as follows : 
I (Mr. Taylor) was called to see a horse said to be suffering from 
disease of the kidneys. I found him suffering from brain pressure ; there 
was the characteristic slow pulse, 36 per minute. He was about thirty 
years old, and had been blind for five years. I got him led with difficulty 
to my infirmary, administered a large dose of physic, and applied cold 
water to the head. 
The bowels acted well in two days, after which I blistered him on the 
poll, but he made no improvement, and I had him killed by cutting the 
carotid artery. I had the brain carefully removed, taking off the base 
of the skull first, which I consider a preferable mode to the usual plan 
of taking off the top first. 
The following is a description of the brain : 
“ The brain itself was healthy, so also the main blood-vessels. The 
dura mater, however, showed, on the right side of the vasal portion, 
considerable thickening, and in some parts a calcareous condition, 
these results being probably due to pachymeningitis. The thickened area 
corresponded to the posterior part of the frontal lobe and a small 
portion of the temporo-sphenoidal lobe on the under surface of the 
right cerebral hemisphere, and extended inwards over the pituitary 
body and optic chiasma; it encroached but slightly to the left of the 
middle line. 
In the midst of the thickened dura mater, and apparently between its 
outer and inner layers, an effusion of blood from one of the meningeal 
vessels had given rise to a large clot, which measured one and a half 
inches from before backwards, and one and a quarter inches at its 
widest part. 
The clot was ol tolerably recent formation, and showed under the 
microscope blood-corpuscles and debris , without any crystals of haema- 
toidine, such as are found in old apoplectic extravasations. 
