Morphology  of  the  ‘Jock'  Trypanosome 
In  the  original  and  the  sub-inoculated  dog  the  parasite  seen 
corresponds  in  every  way  with  that  already  described  in  ‘  Dip  ’  and 
in  '  Balungu  ’ ;  to  this  description  we  have  nothing  to  add. 
Trypanosomiasis  Occurring  in  a  Naturally  Infected  Pig 
On  October  ;th  we  received  from  a  European  in  Abercorn  a 
young  male  pig  for  experimental  purposes.  This  animal  was  one  of 
a  litter  born  in  May  at  Abercorn,  where  within  fifty  miles  Glossina 
morsitans  or  animal  trypanosomiasis  have  never  been  seen  or 
suspected,  and  where  there  is  no  traffic  to  introduce  it.  On  arrival 
this  animal  was  footsore  and  dull,  but  this  was  ascribed  to  the  conduct 
of  the  native  who  drove  it  to  us  a  distance  of  fifty  miles  in  two  days. 
It  was  kept  in  our  camp  and  appeared  quite  healthy  until  October 
31st,  when  the  native  in  charge  reported  it  sick.  On  examination  it 
was  recumbent,  semi-comatose,  breathing  heavily,  visible  membranes 
injected,  consideiable  oedema  of  the  prepuce  and  a  temperature  of 
102°  Trypanosomes  were  swarming  in  the  blood,  and  the  animal 
died  six  hours  later. 
Autopy  commenced  immediately.  Oedema  of  prepuce: 
vessels  markedly  congested ;  body  fat  plentiful. 
all  subcutaneous 
The  same  marked  congestion  in  all  mesenteric  veins:  liver  and  kidney  slightly 
congested.  Spleen  considerably  enlarged,  soft  and  friable,  with  Malpighian  bodies 
prominent.  Some  excess  of  blood-tinged  fluid  in  the  pericardium  ;  coronary  vessels 
of  heart  greatly  distended.  Ante-mortem  clots  in  both  auricles  and  ventricles. 
lUood  dark  and  clots  readily ;  lungs  pale. 
All  lymphatic  glands  swollen,  soft  and  excessively  haemorrhagic. 
The  picture  presented  was  that  of  very  acute  haemorrhagic  septicaemia 
We  are  quite  unable  to  say  where  infection  was  derived  ;  the 
sudden  death  indicated  rapid  disease  due  to  a  local  infection  after 
arrival  in  our  camp.  This  could  not  be  entirely  excluded  owing  to 
existence  of  Stonioxys,  Hematobia  and  Tabanus  in  small  numbers; 
but  the  pig  had  been  in  intimate  association  with  only  Ox  i,  which 
had  an  infection  with  a  trypanosome  (Chunga)  morphologically 
distinct,  and  which  only  became  infected  five  days  prior  to  the  pig’s 
death.  From  the  fact  of  its  indisposition  on  arrival  we  might  surmise 
a  previous  infection  from  some  as  yet  unknown  source  in  Abercorn. 
There  the  pig  had  been  in  intimate  contact  with  sheep,  the  histories 
of  which  are  always  uncertain  owing  to  the  possibility  of  traffic  in 
