REPORTS OF CASES. 
keeping the horse in a dark box stall, light feed. The horse got 
over these acnte symptoms all right, but in general the clinical 
symptoms of chronic hydrocephalus became far more pro¬ 
nounced. During the next few weeks I had occasion to 
watch the animal several times and I saw, when it was going to 
lie down, it would first crouch on its hind legs like a dog and 
then throw himself over sideways, then lying perfectly quiet. 
While eating, the mastication was always performed slowly and 
with long intermissions. Quite frequently the animal would 
stand there for a long time, the head down, the mouth filled up 
with hay, not chewing at all. When not eating, I saw several 
times that the extremities were in the most abnormal position, 
either all four legs gathered under the body or even the front 
extremities crossed. When water was put before the animal it 
would all at once put the head into the pail over the nostrils. 
I advised the owner to have the horse destroyed, as neither 
he nor anybody else could run the risk to sit behind the horse. 
The owner did not seem to like this idea, he still thought I was 
mistaken ; I then told him if my diagnosis was wrong I was 
willing to pay for the horse. We decided to call on two disin¬ 
terested professional men, one being a veterinarian, the other 
a physician, a well-known pathologist. The horse was poisoned 
with cyanide of potash, the brains were carefully taken out ; 
the correctness of my diagnosis was confirmed. 
Post-mortem revealed the following alterations of the brain : 
atrophy of the cortical substance of the hemispheres, consider¬ 
able dilatation of the lateral ventricles and also of the third ven¬ 
tricle, containing about 15 c.ni. of serous liquid. Microscopi¬ 
cally I found principally atrophy of the ganglia cells, on some 
places proliferation of glia cells, on others commencing fatty 
degeneration of the glia substance, desolation of fine blood ves¬ 
sels, infiltration of perivascular lymph sinuses with plenty of 
emigrated leucocytes. 
A CASE OF RUPTURE OF THE FLEXOR METATARSI. 
By J. A. Sloan, Student, McKillip Veterinary College, Chicago. 
A brown stallion, weight about 1000 lbs., was used on a 
newspaper wagon. In his work on December iith he slipped 
on the ice, and when he attempted to rise his near hind foot 
slipped back a great distance. He immediately went lame. 
There was no improvement on the 14th, nor a great deal of 
soreness. An abrasion of the skin was visible just below the 
stifle joint, but was not swollen or sore. 
