16 The Colorado Experiment v Station. 
potassium permanganate 2 per cent and Eugols solution of iodine 
straight, are very useful. This treatment should be employed daily 
until the animal shows considerable improvement. Reports from 
this treatment have been very satisfactory, many saying that after 
the treatment was started they did not lose an animal. 
Prevention consists in isolating all animals infected, and com¬ 
plete disinfection of the barns and corrals with a 5 per cent solu¬ 
tion of crude carbolic acid, six ounces to the gallon. This can be 
best administered with a spray pump. The solution should con¬ 
tain enough lime so that it will be quite apparent when all surfaces 
have been covered with the spray. Some corrals seem to be so badly 
infected that the disease breaks out regularly every year. In such 
places, thorough disinfection is almost a necessity. It is doubtful 
if much can be done by withholding certain kinds of food because 
most any kind of hay is likely to produce abrasions in the mouth. 
It would certainly be wise, however, to feed the best hay to the 
calves, giving that which is coarser and contains thistles to the older 
animals. 
* j 
SORE MOUTH IN PIGS. 
History. 
Sore Mouth or Necrobacillosis of pigs first received general at¬ 
tention in Colorado in the years 1904 and 1905. For the next two 
or three years it reached rather appalling proportions, particularly 
in the San Luis Valley, where it became such a menace to hog rais¬ 
ing that it practically stiffled the industry. During those years it 
became so prevalent in the young pigs about Denver that it was 
causing more trouble than cholera. This meant much when it is 
remembered that cholera was a constant resident in the pens at that 
time. Also it was responsible for the death of many hogs through¬ 
out the Arkansas Valley, in Northern Colorado and along the 
Platte as far east as the state line. 
Unquestionably there was a generous admixture of cholera in 
some of these outbreaks, and it has always been an open question in 
the minds of those who made the investigations as to just how much 
of the loss to attribute to cholera and how much could property be 
charged to the bacillus necrophorous. Many outbreaks showed 
typical necrotic ulcers in the mouth and throughout the alimentary 
canal with many skin and sheath lesions, while others gave the les¬ 
ions which are recognized as typical for cholera. Even this would 
not have complicated matters so much had there net been outbreaks 
in which all gradations from the lesions of pure necrotic stomati- 
