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SOCIETY MEETINGS. 
the two pleural surfaces, visceral and parietal, whereas adhe¬ 
sions are quite common in pleural tuberculosis in cattle. 
Dr. Kelly : I would say that I have never noticed a case of 
adhesion of the lung to the chest wall in case of tuberculosis in 
swine. The tubercular nodules seem to be beneath the parietal 
pleura, causing a bulging at places where the tubercles devel¬ 
oped. The tubercles are usually flattened. I have; never 
noticed them to be pedunculated as in the ox. 
Dr. McCurdy: I think this point is very important. I 
have seen a good many cases where the lung is adherent to the 
thoracic wall and the lung is removed with difficulty, but in 
tuberculosis the lung is not adherent ; it comes out en masse. 
Dr. Forbes: I think this association is indebted to Dr. 
Kelly in bringing this subject to our notice. The prevalence 
of tuberculosis in swine is becoming so frequent that it forces 
itself upon our attention. We are also indebted to him for the 
manner in which he has brought it to notice, having presented 
it in such a graphic and interesting way. He has brought the 
attention of the meeting to the fact that the spleen seems to be 
a primary seat of the disease. The spleen being an accessory 
organ of the digestive system, and the abdominal form of tuber¬ 
culosis being the most frequent in swine, it is easy to account 
for this. It has been noticed in tuberculosis in cattle, that the 
spleen is seldom, if ever affected, the nodules sometimes found 
on the surface being confined to the capsule, and do not pene¬ 
trate the spleen tissue. The difference in the anatomical con¬ 
formation of the two animals would seem to account for this. 
In cattle, the spleen is attached to the greater curvature of the 
rumen, and not in close proximity to the true stomach, while 
in hogs it is almost in direct contact with the true stomach. In 
speaking of phagocytosis, it was stated by the essayist that the 
phagocytes seized hold of the bacilli, and immediately trans¬ 
ported them to the deeper organs, to complete their destruction, 
the spleen being one of the principal organs. During this 
transportation the bacillus may overcome the phagocyte, the 
latter becomes sick, from its effort to digest the microbe, or 
from the effect of the nricrobic secretion upon it, and when 'it 
reaches the spleen the englobed microbe will be able to liberate 
itself, and proceed to form the nodules characteristic of it. An¬ 
other point which interested me was the point brought out by 
Dr. Stewart, regarding the entrance of the bacillus into the tis¬ 
sues. Dr. Kelly, I think, stated in his paper that the phago¬ 
cytes were of two kinds, microphages and macrophages, the 
